Quoted message said:Quoted message said:Another interesting perspective ...
"Another myth propagated by the bodybuilding world, which also began in
the early 80's, is the idea that one must train to failure. Nothing
could be further from the truth. This notion came about when High
Intensity Training (HIT) was being espoused as gospel. Mike Mentzer was
the leader of this movement, however was also insane. In his 1925
classic "Secrets of Strength", Earle Liederman wrote that, "a
strong man never overextends himself in training." Olympic lifters and
powerlifters have never trained to failure, yet always make significant
progress. Dr. Terry Todd, a world powerlifting champion often says if
you are training to failure you are training to fail. Training to
failure does nothing more than over stress the central nervous system
and increase the time it takes to recover from training. Although it
seems hardcore and intense it is generally not a good idea. In fact,
well respected strength coach Chad Waterbury has stated that taking as
little as one set to failure in a particular workout can extend the
recovery process by up to 48 hours. Always end sets with at least one
rep left in the tank."
http://www.strengthcats.com/JFroadtorecovery.htm
What is wrong with training to failure?
Training to failure directly inhibits voluntary strength. Basically,
training to failure fries your nerves (not the technical term ) and
prevents them from being able to contract the muscles for long periods
of time. So when one trains to failure and then waits until strength is
recovered to train a muscle again, oftentimes the muscle has long
recovered and is waiting for the nervous system to catch up.
The alternative to training to failure is HST (hypertrophy-specific
training) which was advocated by Bryan Haycock
http://www.hypertrophy-specific.com/hst_index.html
With HST, you training through soreness. Soreness is not harmful, and
people generally find that training a sore muscle will cause the
soreness to stop.
It is commonly misunderstood that muscle failure is the stimulus for
muscle growth. Unfortunately, this is not true! The tension on the
muscle is what actually causes growth
- Warren GL, Hayes DA, Lowe DA, Armstrong RB. Mechanical factors in the
initiation of eccentric contraction-induced injury in rat soleus
muscle. J Physiol. 1993 May;464:457-75
HST is based on physiologically sound principles not numbers. In short,
they are:
· Progressive load
· Training volume
· Training frequency
· Conditioning (Repeated Bout effect)/Strategic Deconditioning
So we are dealing with 4 basic issues, Load, Volume, Frequency and
Conditioning. Within these basic factors we have reps, sets, and rest.
HST differs from previous training methods in many aspects, but
particularly in how it incorporates knowledge of how the "cell"
physiologically responds to the training stimulus in its methodology.
Previous methods focus on effort (A.K.A Intensity), current voluntary
strength, and psychological factors such as fatigue and variety.
· The number of Reps is determined by the minimum effective load
(this changes over time based on Conditioning)
· The number of Sets is determined by the minimum effective volume
(this changes over time according to current load and Conditioning.)
· The Rest between sets is determined by the amount of time required
to regain sufficient strength to successfully achieve the minimum
effective Volume.
The Frequency (rest between workouts) is determined by the ability of
the CNS to recover sufficiently to maintain baseline "health"
indicators. It is also determined by the time course of genetic
expression resultant from the previous workout.
· The interval of Strategic Deconditioning (SD) is determined by the
time course of adaptation to the individuals maximum weight loads. In
other words, SD is required to reset growth potential after plateauing.
The duration of SD is determined by the level of conditioning attained
during the training cycle.
Mechanical tension on the protein structures of the muscle cells is the
primary stimulus for hypertrophy. This tension can elicit anabolic
processes with or without damaged to the cell membrane. However, some
damage to the cell membrane seems to be critical for the action of
autocrine and paracrine growth factors (FGF, IGF-1, etc). Without the
activity of these growth factors outside the cell there will be no
increase in myonuclei, and thus no significant increase the the volume
and/or number of the cells.
Some improvements in muscle cell function do occur even if the number
of myonuclei remains the same. These won't lead to significant
hypertrophy though. These improvements in muscle cell functional
capacity involve ERK1/2. This is the pathway activated most when you
get an intense burn and/or train to failure.
Muscle "activity" such as the typical repetition, and the metabolic
byproducts and change in the internal millieu of the cell also
"contribute" to hypertrophy, but only indirectly. Reps, and fatigue
activate signaling proteins and transcription factors that increase
protein synthesis. This increase in protein synthesis allows an
increase in crucial enzymes, receptors (yes even androgen receptors),
membrane and structural proteins. Remember that protein breakdown is
also accelerated so the net effect is most often merely a maintenance
of muscle protein levels. This is what goes on after each workout when
plateauing after years of training.
As mentioned, without the activity of IGF-1 and FGF outside of the
cell, satellite cells will not contribute significantly to hypertrophy.
The process is dependant on microtrauma at some degree.
Studies have shown that the ability of a given amount of tension to
elicit hypertrophy decreases over time in a given muscle. This is
because the same adaptive process that leads to muscle growth, also
leads to resistance to the stimulus of muscle growth. It has a lot to
do with the principle of homeostasis, in other words, the body will
always fight further change as it's changing.
Studies have shown too much microtrauma is a bad thing. The rapid
infiltration of immune factors (the primary cause of DOMS) actually
causes significant breakdown of muscle proteins and the death of some
cells.
Now, the ability of mechanical tension to cause microtrauma to the cell
membrane is dependant on the condition of the extracellular matrix. If
it is robust as a result of chronic strain, is will take an
unaccustomed load to induce any trauma. Your ability to apply this load
is dependant on your voluntary strength. Your body is able to protect
your muscle cells from microtrauma even when using max loads. It isn't
always able to protect tendons.
Anytime you do a set and it burns like crazy (painful burn) you are
creating the same conditions of the occlusion studies. In other words,
its not that the effects seen in this study don't happen without
cutting off the blood supply, they do depending on the type of set. I
would guess the vascular occlusion is increasing phosphorylation of
MAPKerk1/2. erk1/2 appears to be more sensitive to acidosis, and oxygen
radicals, both of which would be increased by lack of blood flow.
Although less of a contributor than p38, erk1/2 does appear to
contribute to hypertrophy.
Keep in mind that as a muscle contracts, it squeezes the blood out from
the blood vessels around it. That is why your blood pressure goes up as
large muscle groups contract (even clenching your fists actually raises
blood pressure). This is also why pilots learn to contract their
musculature to keep from passing out during high G-forces.
Contracting and relaxing a muscle acts like a blood-pump and plays a
role in proper function of the cardiovascular system during exercise.
As was mentioned earlier, if you can increase the level of metabolic
byproducts, decrease the pH and increase the level of oxygen radicals
you will "help" to stimulate hypertrophy. However, this is not
sufficient to elicit significant hypertrophy in the absence of
progressive loading. In other words, flexing your muscles until they
really burn won't really make you grow all that well. But combine it
with progressive load and you will facilitate growth.
For more information, go here:
http://www.hypertrophy-specific.com/hst_index.html
http://www.hypertrophy-specific.com/articles.html
http://www.hypertrophy-specific.com/cgi-bi...cgi?act=SF;f=13