Martin said:
Martin Thompson said:11:43:57 Sat, 4 Oct 2003sci.med.nutrition For a simple answer, these are the factors that I can
think of off the top of my head:
1) fewer carbs mean (hopefully) no spikes, so no emergency insulin dumps needed and so
resistance can reduce (as mentioned earlier);
2) proteins convert to glucose, yes, but much more slowly than carbs - hence, again, no spikes
unless you really over-do it;
3) much of the protein you eat is not converted to glucose, but converted to body tissue.
Exercise helps with this;
4) your body can burn fat for energy without the use of insulin and without converting it to
glucose (it burns ketones instead - they can cause a particular type of bad breath too. The
process is called ketosis, and is not to be confused with ketoacidosis, a problem of type 1
diabetics); there is no energy or stamina penalty for burning fat instead of carbs - your
body is perfectly well able to burn either, although carbs are better for a quick burst of
energy should you be feeling depleted at some point, and of course for raising your blood
glucose levels quickly should that be necessary;
5) exercise, assuming you are doing some, burns up glucose stores from muscle cells, without the
use of insulin (the glucose is already in there); subsequent glucose intake replenishes these
stores and keeps your blood glucose levels lower than they might otherwise have been,
preventing the need for insulin spikes and the resulting resistance (it takes a couple of
days to replenish the stores after an average workout, usually);
6) burning fat reduces insulin resistance for another reason: fat can physically block the
insulin receptors on cells;
Quoted message said:Thanks Martin, that was what I needed to know, especially 4). I understood that consuming fats
did not immediately raise glucose levels but was under the impression that fats went to the liver
and were eventually converted to glucose in order to be burnt, in which case insulin would still
be needed.
Regarding 4): When fat is being used for energy the triglycerides stored in fat cells are broken
down and the fatty acids that are released enter the blood stream, where they can be taken up by any
cells that want them. Alternatively, the liver can release fat bundled with protein carriers (VLDL,
LDL etc.) and the fatty acids will be removed as required by the rest of the body. Ketone bodies are
normally a small contribution to fat burning, but increase greatly when the body has been deprived
of food for a prolonged period (starvation). Fatty acids don't get converted to glucose, nor do
ketones and insulin is never required for burning glucose, only storing it.
The liver can take up amino acids without insulin and may convert these to glucose, particularly if
blood glucose levels have dropped. If this is the case it means the brain is likely to be short of
glucose (it can't burn fatty acids like the rest of the body). The whole reason blood glucose levels
are controlled is for the brain's requirements. The brain can take up glucose without the need for
insulin, so the only reason insulin is released is when glucose levels get too high and need to be
reduced (by pushing the glucose into other cells to be stored). Interestingly, *exercising* muscles
can also take up glucose without the need for insulin.
Your body want blood glucose levels constant for feeding the brain, but every time you eat carbs
they get messed up and insulin is the body's emergency response. Ideally most of the body will run
on fats, with glucose reserved for the brain, red blood cells and one or two other cell types. By
going low carb the blood glucose levels will rarely get high enough to trigger significant insulin
release, so the only tissues burning glucose are the ones that don't need insulin to remove it from
the blood.
MattLB