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Death By Enrichment

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  1. Title 39 - Trade and Commerce

    http://www.scstatehouse.net/code/t39c027.htm

    CHAPTER 27.

    FLOUR AND BREAD

    SECTION 39-27-10. Enrichment of certain flour.

    It shall be unlawful for any person, except as hereinafter provided, to
    manufacture, mix, compound or sell for human consumption in this State
    any white flour or self-rising flour made from wheat which does not
    contain the vitamins and other ingredients contained in the following
    provisions:

    (1) White flour shall contain in each pound not less than one and
    sixty-six one-hundredths milligrams and not more than two and five
    tenths milligrams of vitamin B-1 (thiamine), not less than six
    milligrams and not more than twenty-four milligrams of nicotinic acid
    (also recognized under the name of niacin) or nicotinic acid amide
    (also known under the name of niacin amide) and not less than six
    milligrams and not more than twenty-four milligrams of iron (Fe);

    (2) If other vitamins or minerals are added to bread or flour, they
    shall be added only in accordance with the regulations of the
    Department of Health, Education and Welfare;

    (3) These ingredients and amounts are in accordance with the definition
    of enriched flour as promulgated by the Federal Security Agency
    (Federal Register, May 27, 1941 pp. 2574 to 2582, and December 3, 1941
    pp. 6175 and 6176, postponing the effective date of riboflavin as a
    required ingredient in enriched flour) and the State Department of
    Agriculture shall change or add to the specifications for ingredients
    and the amounts thereof as necessary to conform to changes in the
    Federal definition of enriched flour; and

    (4) Iron shall be added only in forms which are harmless and
    assimilable.

    <My Comment>
    THE HELL WITH THE FORM, WHAT ABOUT THE QUANTITY!
    </My Comment>

    http://www.ironoverload.org/hippocrates.html

    Hippocrates Magazine

    November/December 1995

    Page 28

    "The Shocking Truth About Iron"

    A Million Americans have a potentially fatal disease--iron overload. So
    why are we still focused on deficiency?

    by Steven Finch

    The oft-repeated first law of modern medical diagnosis is that when you
    hear hoofbeats, think horses not zebras. So in 1951, when 19-year old
    Roberta Crawford complained of constant fatigue and lingering flu
    symptoms, her doctor took one look at her pale skin and thin frame and
    heard the clipperty-clop of iron deficiency anemia. He sent her off
    with a prescription for a new iron supplement called Trinsicon. "At
    first it seemed to help," Crawford recalls, "but then I'd get clobbered
    by the flu again, only worse." She started with a single capsule each
    day, but when her doctors noted that the Trinsicon wasn't curbing her
    anemia, they bolstered the dose. by the time Crawford was up to five
    pills a day, she'd become disabled from sheer exhaustion. "I knew I
    didn't want the iron," she says, "but when I asked one doctor if I
    could give it up, he said, 'No. It's crucial for your life. You have to
    keep taking it forever.'"

    Crawford took iron for her "deficiency" for the next 27 years. Then in
    1978, Crawford's swollen liver led her gastroenterologist to make a
    lifesaving diagnosis. To the GI, a distended liver, coupled with her
    symptoms of fatigue and flu, sounded a lot like cancer. He ordered a
    biopsy. The liver sample came back clean, except that it was brimming
    with iron. He then ordered a series of blood tests, including one to
    determine her levels of the iron-binding protein, ferritin. Healthy
    adults have up to 160 grams in a deciliter of blood. A measure of 300
    grams can indicate advanced iron overload. Roberta Crawford's ferritin
    approached 7,000. "You have an interesting, rare disease," her doctor
    told her. "Hemochromatosis".

    The GI was right about the diagnosis, but he was off the mark about its
    frequency. A hereditary disease in which the intestine absorbs excess
    iron, hemochromatosis is, in fact, the most common and one of the most
    misunderstood--and most underdiagnosed--genetic disorders in the
    country. The hemochromatosis gene is 30 times more prevalent than the
    gene for Duchenne muscular dystrophy and occurs far more frequently
    than the genes for either Down syndrome, cerebral palsy, or cystic
    fibrosis. All told, more than a million Americans (about one out of
    every 250) suffer from hemochromatosis. And that's not counting the
    gene in the Americans (fully 23 million) who are carriers of the genes
    for the disease.

    Normally, healthy people absorb about 10 percent of the iron in the
    foods they eat. Carriers, who have only one of the two hemochromatosis
    genes, can absorb half-again that much, while Crawford and others with
    full-blown hemochromatosis can soak in up to 20 percent or more. Since
    the body has no natural way of excreting iron (other than through a
    woman's menses and the minuscule amounts everyone sloughs off in dead
    skin), any extra iron that's absorbed is relentlessly dumped into
    tissues and vital organs, especially the heart, liver, and pancreas.
    The good news is that, if hemochromatosis is discovered early,
    phlebotomies, or bloodletting, will entirely prevent illness associated
    with the disease. But left unchecked, excess iron can turn the skin a
    coppery, bronze, or grayish color and clog vital internal organs to
    such an extent that initial, vague ailments such as stomach pain and
    fatigue can develop into arthritis, diabetes, cancer, heart disease,
    cirrhosis of the liver, and a host of other ills.

    Unfortunately, iron overload is left unchecked all too often. Surveys
    of hemochromatosis patients conducted in 1985 and 1988 by the Iron
    Overload Diseases Association found that the average patient had to
    wait more than five years before being properly diagnosed. Most of
    those surveyed had consulted multiple doctors; 25 percent of them also
    had a type of anemia unrelated to iron status and, like Crawford, were
    actually first prescribed iron before anyone successfully diagnosed
    their hemochromatosis.

    Given iron overload's prevalence and seriousness, why the long delay?
    For one thing, say a group of health experts who are trying to raise
    physicians' awareness, symptoms, such as fatigue and pain in the
    joints, are fairly nonspecific. For another, our decades-old
    preoccupation with iron deficiency anemia has too often led medical
    schools--and therefore the medical profession--to focus attention on
    the benefits of iron at the expense of its dangers.

    Even today, says Kenneth Bridges, an assistant professor of medicine at
    Harvard who has spent the last 15 years researching iron's effects on
    the body, most doctors aren't taught enough. "A lot of medical schools
    still only skim over iron deficiency and don't even touch on
    hemochromatosis except to say that it's a rare iron disease that
    yellows the skin," he says. As a result, despite more than a million
    people at serious risk, hemochromatosis remains an invisible problem.
    Lack of physician awareness, Bridges says, extends even to his own
    clinical affiliation, Harvard's prestigious Brigham and Women's
    Hospital in Boston.

    Iron is, of course, essential to life. Skimp on iron and the result is
    anemia, a condition that decreases hemoglobin in the blood, which in
    turn, cheats the body of enough oxygen for proper growth and
    functioning. Sic percent of Americans--almost exclusively infants,
    adolescents, and women in their childbearing years--need additional
    iron.

    It's known, for example, that while adequate iron is vital to healthy
    pregnancies, about 20 percent of women of child-bearing age are iron
    deficient. Numerous studies have confirmed that low iron stores in
    children can impair physical and mental development. That's an alarming
    fact when you consider that, according to a 1994 Johns Hopkins study,
    at least 15 percent of all adolescent girls have some form of iron
    deficiency.

    But as a group, Americans are in much more danger from overloading on
    iron than from getting too little. For one thing, although iron
    deficiency is the most common reason for anemia, it's far from being
    the only one. Anemias are caused by everything from vitamin
    deficiencies to defects in the hemoglobin itself or in the bone
    marrow's production of red blood cells. That distinction isn't always
    clearly understood. Like Crawford (whose anemia turned out to stem from
    insufficient vitamin B-12), many anemic patients are prescribed iron as
    a matter of course, sometimes with drastic consequences.

    William Crosby, a former director of hematology at the Chapman Cancer
    Center in Joplin, Missouri, calls iron deficiency anemia a "nothing
    problem"--a statement that puts him at odds with many in the medical
    community.

    "What I mean is that although a lot of women have iron deficiency
    anemia and are worn down and fatigued by it, their deficiency doesn't
    kill them, " says the 30-year military veteran, who introduced
    hematology as a specialty to the Army Medical Corps. "Iron overload is
    lethal."

    Hematologist Victor Herbert, a professor of medicine at Mount Sinai and
    Bronx Veteran's Affairs Medical Centers in New York City, and one of
    the world's foremost experts on nutrition, echoes Crosby's concern.
    Except for high-risk groups like children and premenopausal women, it's
    pretty tough for other Americans to come up iron deficient, he says.
    Not only is iron abundant in unprocessed meat and vegetables, but since
    the early 1940's the FDA has required that virtually all four products
    be iron- enriched. so while in the rest of the world more than a
    billion people are iron-deficient, in this country you can't trip over
    a Twinkie without getting metal shavings on your sneakers. In fact,
    Americans who need extra iron are outnumbered nearly 20 to one by those
    who don't. Nevertheless, tens of millions of Americans regularly
    supplement their diets with multivitamins, minerals, or other iron
    products because they believe it will keep them healthier.

    Worse, doctors frequently prescribe iron without fully diagnosing the
    cause of a patient's anemia or ruling out iron overload. "It happens
    all the time," says Herbert. "And that's just plain dangerous. No one
    should take a supplement without having their iron status checked."
    Most hemochromatosis carriers, for example, will live full, healthy
    lives despite absorbing twice as much iron as the average person. "But
    give carriers a bunch of multivitamins or a string of iron supplements
    like Geritol," says Herbert, "and you're going to throw them into iron
    overload too." The bottom line, he adds, is that people who take iron
    are twice as likely to be hurt by a supplement as they are to be helped
    by it.

    What's needed, Herbert and his colleagues maintain, is a sharp increase
    in doctors' awareness of how serious a problem hemochromatosis is as
    well as universal screening for patients' iron status. The old
    definition for hemochromatosis won't do anymore, says David Witte, a
    University of Iowa pathologist who's written new guidelines on
    hemochromatosis due out sometime in early 1996. (See additional
    information attached to this article on how to order a copy of these
    guidelines).

    "We have to move beyond thinking about this disease as a liver malady
    in old men," Witte says. "For one thing, the condition affects as many
    women as men. Women just have an early advantage, which they lose after
    menopause." For another, he adds, we're doing patients an inexcusable
    disservice by refusing to recognize hemochromatosis as a chronic,
    genetic disorder that makes people absorb excess iron. Using this new
    definition, he says, the challenge becomes to find these people and
    treat them before their excess iron causes complications.

    "If you don't look for hemochromatosis until a patient displays
    clinical symptoms or until a liver biopsy turns up full of iron,"
    Bridges says, "too much damage has already been done."

    Screening would go a long way in reducing such damage, Witte and other
    experts insist. "People are getting bounced around from doctor to
    doctor, suffering from fatigue or headaches, and no one thinks to check
    for iron overload," Witte says. "A few patients are still being sent to
    psychiatrists, because their doctors pass them off as hypochondriacs."

    Roberta Crawford remembers when doctors told her she was crazy. Now 63
    and quite sane, Crawford's only concessions to iron overload are
    phlebotomies once or twice a year and regular yoga and swimming to ease
    her nagging arthritis. But she's come to realize how lucky she is to
    have survived her condition relatively unscathed. Since starting the
    Iron Overload Diseases Association in 1981, Crawford has met or talked
    to the families of hundreds of seemingly healthy people--many who'd
    never taken an iron supplement--who became permanently disabled or died
    in their thirties or forties, a few even younger. Crawford and her
    association have been trying to educate both doctors and the public
    alike about the need to know their iron status. A second goal is to
    convince public health officials that it's worth the cost to screen
    seemingly healthy patients for a hidden disease.

    Managed care giant Kaiser Permanente may already be convinced. At
    Kaiser San Diego, home of the country's single largest preventive
    medicine program, doctors in its department of preventive medicine are
    testing the iron status of every patient they see (until they reach
    30,000). So far, of the approximately 8,000 patients they've tested,
    about one out of every 250 has had hemochromatosis. Most have been
    treated. That's fine for a relatively few southern California patients,
    says Bridges, but until screening becomes universal, it's up to
    individual practitioners to decide. "Look at it this way," he says.
    "There just aren't many diseases out there that a million Americans
    have. And while there's nothing you and I can do about advanced colon
    cancer or diabetic end stage renal disease, we can save a whole lot of
    people from hemochromatosis.

    Appendix

    Iron Overload Diseases Association, Inc. (IOD)
    433 Westwind Drive
    North Palm Beach FL 33408-5123 561-840-8512 fax
    561-842-9881

    "Practice Parameter for Hereditary Hemochromatosis," David L. Witte et
    al., in press, College of American Pathologists, Northfield, Illinois.

    "Diagnosis and Treatment of Iron Disorders," Victor Herbert et al.,
    Hospital Practice Symposium Supplement, Vol. 26, Supplement 3, 1991.

    The Iron Elephant: What You Should Know About the Dangers of Excess
    Body Iron, Roberta Crawford, Glyndon, Maryland: Vida Publishing, Inc.
    1992.

    tick ... tick ... tick ... by Roberta Crawford (a novel)
    "Spellbinding, moving mystery," says Clive Cussler, best selling
    novelist.

    National Heart Lung and Blood Institute Information Center, Bethesda,
    Maryland.

    -------------------------------------------------------------------------------------------------------------
    FERROUS FABLES: EIGHT MYTHS ABOUT IRON

    Anyone whose family had a television probably grew up watching
    Popeye-the pipe-smoking, pip-squeak of a sailor who could transform his
    sagging biceps into twin turbines with a few gulps of iron-rich
    spinach. Well, Popeye lied. Extra iron, whether from spinach or
    supplements, doesn't make us any stronger, smarter, faster, or more
    attractive. Here are some other common ironclad misconceptions.

    Iron supplements are not good medicine. Almost certainly not for most
    men--who have no natural way of getting rid of excess iron--and not for
    women.

    An anemic woman is not always iron deficient. She could be, but iron
    deficiency is only one from a long list of underlying problems that can
    trigger anemia, including vitamin deficiencies, defective hemoglobin,
    and even iron overload.

    An enlarged liver means your patient drinks too much? Not necessarily.
    Sure, some patients will flat out lie about how much alcohol they
    drink. But if you don't know why Mr. Jones' liver is enlarged; and he
    insists he's a teetotaler, screen for iron.

    Women can also get too much iron. The genes for hemochromatosis are
    equally distributed between genders. Tthey are not at lesser risk,
    women with hemochromatosis, even those in their childbearing years, can
    easily absorb more iron than is healthy.

    You can't cure hemochromatosis by eating less iron. It's impossible to
    avoid iron in the American diet. Iron resides in every living plant and
    animal cell, and virtually all commercials flours are iron-enriched.

    Switching to a multivitamin without iron will help. Only marginally.
    Most multivitamins contain small amounts of elemental iron. More
    important, vitamin C enhances the body's ability to absorb iron.

    <My Comment>
    RIGHT, AND GET LOTS OF VITAMIN C.
    </My Comment>

    http://groups.google.com/group/alt.conspiracy?lnk=li&hl=en
    http://www.google.com/search?hl=en&q=conspiracy+links&btnG=Google+Search

    *
    *

  2. Death by spam.

    I.P.


  3. Quoted message said:

    Death by spam.

    http://www.spam.com/sp.htm

    http://www.ironoverload.org/facts.html

    14. Excess iron lowers the immune system. Many diseases will show a
    poor outcome unless any excess iron is removed: AIDs, cancer and
    hepatitis, for example.

    15. Iron does cross the blood brain barrier, contrary to old belief.
    Excess iron stored in the brain has been found to exacerbate severity
    in Alzheimer's, MS, Lou Gehrig's, Parkinson's and other diseases. Iron
    in the brain also leads to psychological problems.

    Quoted message said:


    I.P.2.

  4. Quoted message said:
    Quoted message said:

    (4) Iron shall be added only in forms which are harmless and


    assimilable<<

    'harmless' .. may be the operative word ..

    Since it has been recommended those with tuberculosis NOT consume iron
    high foods .. and since one third of the world is already infected ..
    then the addition of iron TO our .. foods .. may NOW .. be ..
    considered NOT .. to BE .. 'harmless' .. ?

    Dietary Iron Associated With Pulmonary Tuberculosis In Rural Africans
    A DGReview of :"Association of Pulmonary Tuberculosis with Increased
    Dietary Iron"
    Journal of Infectious Diseases

    10/17/2001
    By James Adams

    Increased dietary iron is associated with a 3.5 fold increase in the
    estimated odds of developing active tuberculosis in rural Africans.

    Researchers from multiple institutions, including the University of
    Zimbabwe School of Medicine in Harare, Zimbabwe, and the National
    Institute of Child Health and Human Development (NICHD) in Bethesda,
    Maryland, United States, investigated the possibility that increased
    dietary iron is a risk factor for tuberculosis.

    "Exposure to high levels of dietary iron in the form of traditional
    beer is associated with increased iron stores in rural Africans,"
    according to the investigators.

    Ninety-eight pulmonary tuberculosis patients and 98 controls from rural
    Zimbabwe were included in the study. Their dietary iron history and
    human immunodeficiency virus (HIV) status were evaluated.

    Results showed that HIV seropositivity was associated with a 17.3-fold
    increase in the estimated odds of developing active tuberculosis. Also,
    HIV seropositivity in patients treated for tuberculosis was associated
    with a 3.8-fold increase in the estimated hazard ratio of death.

    Increased dietary iron was associated with a 3.5-fold increase in the
    odds of developing active tuberculosis and a 1.3-fold increase in the
    estimated hazard ratio of death.

    The study was financially supported by the Office of Minority Health to
    the Cell Biology and Metabolism Branch of the NICHD, the Cell Biology
    and Metabolism Branch of the NICHD and the J.F. Kapnek Charitable Trust
    in Harare, Zimbabwe.
    J Infect Dis 2001; 184: 936-939. "Association of Pulmonary Tuberculosis
    with Increased Dietary Iron"

    Tuberculosis (Edinb). 2004;84(1-2):110-30. Related Articles, Links

    Iron, mycobacteria and tuberculosis.

    Ratledge C.

    Department of Biological Sciences, University of Hull, Hull HU6 7RX,
    UK. [email hidden]

    The role of iron in the growth and metabolism of M. tuberculosis and
    other mycobacteria is discussed in relation to the acquisiton of iron
    from host sources, such as transferrin, lactoferrin and ferritin, and
    its subsequent assimilation and utilization by the bacteria. Key
    components involved in the acquisition of iron (as ferric ion) and its
    initial transport into the mycobacterial cell are extracellular iron
    binding agents (siderophores) which, in pathogenic mycobacteria, are
    the carboxymycobactins and, in saprophytic mycobacteria, are the
    exochelins. In both cases, iron may be transferred to an
    intra-envelope, short-term storage molecule, mycobactin. For transport
    across the cell membrane, a reductase is used which converts
    FeIII-mycobactin to the FeII form. The ferrous ion, possibly complexed
    with salicylic acid, is then shuttled across the membrane either for
    direct incorporation into various porphyrins and apoproteins or, for
    storage of iron within the bacterial cytoplasm, bacterioferritin. The
    overall process of iron acquisition and its utilization is under very
    genetic tight control. The importance of iron in the virulence of
    mycobacteria is discussed in relationship to the development of
    tuberculosis. The management of dietary iron can therefore be
    influential in aiding the outcome of this disease. The role of the old
    anti-TB compound, p-aminosalicylate (PAS), is discussed in its action
    as an inhibitor of iron assimilation, together with the prospects of
    being able to synthesize further selective inhibitors of iron
    metabolism that may be useful as future chemotherapeutic agents.

    Publication Types:
    Review
    Review, Tutorial

    PMID: 14670352 [PubMed - indexed for MEDLINE]

    --------------------------------------------------------------------------------

    FEMS Immunology and Medical Microbiology
    Volume 45, Issue 2 , 1 August 2005, Pages 103-112

    doi:10.1016/j.femsim.2005.02.007
    Copyright © 2005 Federation of European Microbiological Societies
    Published by Elsevier B.V.

    Iron and iron chelating agents modulate Mycobacterium tuberculosis
    growth and monocyte-macrophage viability and effector functions

    Leandra Cronjéa, Nicole Edmondsona, b, Kathleen D. Eisenachb and Liza
    Bornmana, ,

    aDepartment of Biochemistry, University of Johannesburg, P.O. Box 524,
    Auckland Park, Johannesburg 2006, South Africa
    bDepartment of Pathology and Microbiology/Immunology, University of
    Arkansas for Medical Sciences, Little Rock, Arkansas, USA

    Received 20 August 2004; revised 6 January 2005; accepted 18 February
    2005. Available online 30 March 2005.

    Abstract
    Excess of iron promotes Mycobacterium tuberculosis infection, its
    replication and progression to clinical disease and death from
    tuberculosis. Chelation of iron may reduce M. tuberculosis replication,
    restore host defence mechanisms and it could constitute an application
    in the prevention and treatment strategies where both iron overload and
    tuberculosis are prevalent. We investigated the effect of iron and iron
    chelating agents, like desferrioxamine and silybin, individually and in
    combination with iron on mycobacterial number, viability in culture and
    after recovery from monocyte-macrophages, together with
    monocyte-macrophages viability and oxidative defence. Mycobacterial
    number and viability in culture were assessed using real-time
    quantitative PCR of H37Rv IS6110 DNA, 16S rRNA and 85B mRNA, whereas
    the microplate AlamarBlueTM assay was used to detect viability in
    culture post-infection. Mitochondrial membrane potential and
    phosphatidyl serine exposure of monocyte-macrophages, detected using
    Mitotracker Red fluorescence and Annexin V binding, respectively,
    served as indicators of host cell viability. Superoxide generation
    served as marker of monocyte-macrophage effector functions.
    Extracellular H37Rv showed a significant increase in number and
    viability in presence of excess iron and, by large, a significant
    decrease in number and viability in presence of the iron chelating
    agents, silybin and desferrioxamine, compared to cultivation without
    supplementation. Intracellularly, excess iron increased H37Rv viability
    significantly but reduced monocyte-macrophages mitochondrial membrane
    potential and compromised superoxide production. Desferrioxamine had
    little influence on intracellular parameters, but consistently
    prevented effects of excess iron, while silybin significantly altered
    most intracellular parameters and mostly failed to prevent effects of
    excess iron. These findings suggest that chelation therapy should be
    considered in conditions of iron overload and that effective chelating
    agents like desferrioxamine, with limited intracellular access might
    need to be used in combination with lypophilic chelating agents.

    Keywords: Desferrioxamine; Silybin; H37Rv; Mycobacterium tuberculosis;
    Iron chelation

    Corresponding author. Tel.: + 27 11 489 2406; fax: + 27 11 489 2605

    FEMS Immunology and Medical Microbiology
    Volume 45, Issue 2 , 1 August 2005, Pages 103-112

    Copyright © 2005 Elsevier B.V. All rights reserved. ScienceDirect® is a
    registered trademark of Elsevier B.V.

    Who loves ya.
    Tom

    Jesus Was A Vegetarian!
    http://jesuswasavegetarian.7h.com

    Man Is A Herbivore!
    http://pages.ivillage.com/ironjustice/manisaherbivore

    DEAD PEOPLE WALKING
    http://pages.ivillage.com/ironjustice/deadpeoplewalking

  5. WOW! Great info; thanks.

    http://members.tripod.com/ironjustice-ivil/manisaherbivore/index.html

    The appearance of iron .. free iron in the body has the same result as
    the iron which is given to you by your doctor in order to correct your
    anemia.

    Your doctor either infuses / injects iron into your vein or he gives
    you iron supplements which raise the levels of iron.

    The appearance of iron .. free iron .. signals the body to produce red
    blood cells .. which in turn the body does exactly what it has been
    told to do .. increase production of red blood cells.

    This is called erythrocytosis .. increased production of erythrocytes
    ... red blood cells.

    The medical profession has quite a large window in which it will either
    treat or not treat what they consider to be increased red blood cells.

    In fact studies have shown the amount of red blood cells considered and
    shown to be detrimental in women .. decreased live birth / pregnancy
    complications .. is well below the cutoff marker used by doctors to
    even consider the fact they are to be treated for excessive red blood
    cell mass / too many red blood cells.

    Too many red blood cells leads to increased red blood cell destruction
    ... and this leads to a buildup of iron in the spleen and kidneys and
    other organs.

    There are many theories as to the cause of disease and this is one.

    The iron from the meat we eat builds to toxic levels in the body and
    causes all disease.

    There are many ways this is manifested , be it the 'feeding' of cancer
    , bacteria or virus' , or the lack of antioxidants in the body for the
    very basics of human immune response.

    Since elevated iron in the body leads to massive levels of RUST /
    oxidation one would conclude either the removal of said iron or the
    attack of the rust produced by the iron would be in order?

    BUT I'M .. ANEMIC .. ?

    Here you will find a substance which has been shown to be effective in
    the removal and the quenching of the rust occuring as a result of the
    elevated levels of iron in the body .

    You will also find studies which support the use of this substance and
    these studies have been conducted by the greatest minds of the day and
    the articles are to be found in the greatest medical database in the
    world.

    In the mouse .. the animal most likely to be used in studies .. they
    have shown when the mouse receives too little phytic acid in his diet
    he has an increase of iron levels in the brain. MANY times the amount
    of iron in the brain than when he receives adequate phytic acid.

    Phytic acid is a substance found in our plant foods .. chiefly our
    grains which is removed in the modern refining of our flours.

    Coupled with an increase of iron this leads to increased oxidation /
    rust in the body which destroys the antioxidants in the body .. much
    needed antioxidants.

    Tocopherol / vitamin E by virtue of BEING an antioxidant is destroyed
    in this increased rust / oxidation .. scenario.

    This leads to many different problems but in this instance we are
    speaking to anemia and thusly appropriately enough .. iron destroys
    tocopherol.

    Hemolysis of red blood cells destroys tocopherol and this leads to a
    circle of increased iron absorption to compensate for the loss of the
    red blood cells. .. PLUS the vitamin E deficiency leads to a poorly
    formed / nonviable red blood cell.

    This poorly formed red blood cell has no staying power and thusly the
    destruction .. early destruction of red blood cell leads to even more
    rust / oxidation caused by the appearance of the iron core of the red
    blood cell .

    This would normally be counteracted by the levels of 'anti' - oxidants
    , the antirust nutrients in the body.

    As previously stated though the lack of tocopherol leads to an almost
    pure state of erythrocytosis / increased production and destruction of
    red blood cells which leads to an iron LOADING hemolytic anemia.

    Thiamin .. an iron binder .. when deprived from an animal results in a
    PURE ERYTHROCYTOSIS .. pure hemolytic anemia .. increased production
    and destruction of red blood cells.

    Thiamin is a B vitamin and all B vitamins are considered to be
    antioxidant and thusly the potential is there for increases in rust /
    oxidation to destroy thiamin and result in increased production and
    destruction of red blood cells leading to hemolytic anemia.

    An anemia of the BLOOD / lack of red blood cells .. but .. NOT an
    anemia of lack of iron.

    The same anemic condition experienced by astronauts.

    Iron is the regulator of red blood cell production.

    When iron appears in serum the body recognizes it and begins its'
    process of recycling what it thinks / believes to be a destroyed red
    blood cell.

    It creates a NEW red blood cell.

    When the body produces TOO MANY red blood cells it is called
    erythrocytosis.

    I have placed this page as a means for a person to conveniently begin
    the steps one needs to take to regain health or offset the progressive
    destruction caused by elevated iron levels in the body.

    Elevation of iron levels lead to problems easily seen by medical
    researchers. These elevated levels of iron many times are not fully
    rendered safe until they are fully depleted as evidenced in medical
    studies.

    Therefore the level of iron in your body which your doctor most likely
    considers safe has been shown in many studies to be actually causing
    problems.

    Studies have shown definite associations between elevated iron levels
    and almost all disease if one considers iron is a prime producer of
    rust / oxidation / free radicals / reactive oxygen species. This rust
    has been considered by many of the greatest minds to be the underlying
    cause to be very basis of human processes .. aging.

  6. What's so funny about peace, love and "I. P. Freely"
    <[email hidden]> posting the following on Sat, 17 Sep
    2005 18:22:42 -0700 iin alt.conspiracy?

    Quoted message said:

    And this relates to prostate cancer and politics HOW?
    Get outta here, dimwits!

    You quoted over 8,000 lines to post this?

    Get off the internet until your brain finishes forming.
    --

    Douglas E. Berry Do the OBVIOUS thing to send e-mail
    Atheist #2147, Atheist Vet #5

    "Men never do evil so completely and cheerfully as
    when they do it from religious conviction."
    Blaise Pascal (1623-1662), Pense'es, #894.

  7. "Douglas Berry" wrote

    Quoted message said:

    What's so funny about peace, love and "I. P. Freely"
    <[email hidden]> posting the following on Sat, 17 Sep
    2005 18:22:42 -0700 iin alt.conspiracy?

    Quoted message said:

    And this relates to prostate cancer and politics HOW?
    Get outta here, dimwits!

    You quoted over 8,000 lines to post this?

    Get off the internet until your brain finishes forming.

    We asked politely for you cross-posters to go away. You didn't listen.

    I've got tears rolling down my cheeks at the 8,000 lines tally. I didn't
    know a few clicks on Google under "iron" had actually hit 8,000 lines; it
    worked better than I thought! You got my point, though, didn't you? Quit yer
    cross-posting to the prostate cancer forum and we'll leave you alone. People
    with cancer, especially terminal cases, have little patience or sympathy
    with irrelevant cross-posters. If ya can't stand the heat, get out of our
    kitchen. The price of admission to our forum is daunting; get back to us
    when your PSA hits 10 and maybe we'll care about your complaint. In the
    meantime . . . hope you guys are on a dial-up connection.

    Conspiracies and politics, my ass.

    I.P.

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