[email hidden] wrote in message ...
Quoted message said:Is fasting really that dangerous for a type I?
What problems can befall a fasting type I if they follow
the following rules?
1) Take glucose as needed to maintain BG levels
2) Keep well hydrated and take proper electrolytes and
vitamins
3) Test often
I'm aware that ketoacidosis is the main concern, but as
long as BG is good and electrolytes/hydration is fine, how
much more dangerous is it than skipping a few meals? I'm
talking about a 1-2 week fast, not a hunger strike.
Dan
Ketoacidosis is caused by a shortage of insulin not a
shortage of food.
If you fast, your body will convert fat and muscle into
energy sources.
I'm fairly certain that you will generate ketones but that
is not the same as ketoacidosis.
This will give you a golden opportunity to totally fine
tune your basal insulin doses. Of course, if your basal is
not fairly well tuned before you start, expect some
problems. If you don't understand your basal now, you will
after your fast. (I don't think many T1 really understand
their basal routines)
Stock up on strips, test often, keep good records,
understand your basal insulin activity profile.
Umm. . .make certain somebody is checking on your safety,
especially if you are using NPH as a basal.
Be conversant with the symptoms of DKA, e.g.
embbs.comdiagn.htmlOpen ↗
"History and Physical:
Patients with DKA usually present with complaint of fatigue,
malaise, thirst, and polyuria. Depending on the length of
symptoms the patient may be able to report weight loss. As
the patient becomes increasingly ill they may begin to vomit
and complain of abdominal pain. The exact cause of abdominal
pain that is associated with DKA is not known. The abdominal
pain is disturbing since it may be secondary to the DKA, or
be from the pathologic process that initiated the crisis,
such as pyelonephritis, pancreatitis, etc. Usually,
abdominal pain secondary to DKA will begin to resolve with
tre atment.
The physical signs of DKA can be variable. Most patients
will have some degree of tachycardia, but the blood pressure
is often normal. Evidence of dehydration, such as loss of
skin turgor, and dry mucus membranes may be present. The
patient may be febrile, and extreme elevations of
temperature should not be assumed to be the result of
dehydration. Hypothermia may also be seen. . ."
Regards
Old Al