Re: Diabetes and Glycogen?

convicted neighbor Kumar said:

Andrew said:

neighbor Kumar said:

Andrew, in the Holy Spirit, boldly wrote:
> neighbor Kumar wrote:
> > Andrew, in the Holy Spirit, boldly wrote:

snip> > > > Whether VATs are formed independetly of total fats status
or above
> > > > SAT?
>
> > > In excess of SAT stores.
>
> > In pre-VAT formation state on exhausting SAT stores capacity, will
> > there be some kind of resistance or IR to furthur SAT stores?
>
> No. The IR arises as a consequence of VAT and not SAT.

Can it be possible that IR starts on start of VAT formation, when SAT
stores are exausted and increases on increase in VAT?

IR starts because of harmful inflammatory cytokines from VAT.

Start of of harmful inflammatory cytokines starts with start of VAT
forming and start of VAT forming will be on exhausting of SAT sores?

VAT starts forming when there is overeating.

Quoted message said:

Quoted message said:

Quoted message said:

It looks, VAT are formed may be for ugent/immediate energy need, if
SAT's breakdown somehow are resisted to be used probably due to more
and continual insulin?

Incorrect.

Though it is unclear but immediate enegy availability from VAT is
indicative in some sistes. Probably, VAT formation may occur in cases
of breakdown of enegy stores are resisted?

VAT formation occurs when there is overeating.

Quoted message said:

Quoted message said:

Quoted message said:

Though VAT may be considered pathological but
still may have some purpose?

The purpose is to cause pathology... to cause harm.

Can't there be some pathology but more benefit for survival--immediate
available of energy on need if other energy stores are restricted/
resisted?

Such is the purpose of glycogen and not VAT.

Quoted message said:

Quoted message said:

May GOD bless you.

Thanks.

You are welcome.

Have redirected your thanks to GOD so we will both be blessed.

Prayerfully in Jesus' ever-lasting love,

Andrew <><
--
Andrew B. Chung, MD/PhD
http://EmoryCardiology.com

May HIS immortal brethren pray for our dying mortal friends and
neighbors:
http://HeartMDPhD.com/Convicts

Especially dear Bob(this one) Pastorio:
http://bobs-amanuensis.livejournal.com/4211.html
http://pics.livejournal.com/bobs_amanuensis/pic/0000z24f/g1

MD/PhD said:

convicted neighbor Kumar said:

Andrew said:

neighbor Kumar wrote:
> Andrew, in the Holy Spirit, boldly wrote:
> > neighbor Kumar wrote:
> > > Andrew, in the Holy Spirit, boldly wrote:

Quoted message said:

Quoted message said:

> snip> > > > Whether VATs are formed independetly of total fats status
> or above
> > > > > SAT?

Quoted message said:

Quoted message said:

> > > > In excess of SAT stores.

Quoted message said:

Quoted message said:

> > > In pre-VAT formation state on exhausting SAT stores capacity, will
> > > there be some kind of resistance or IR to furthur SAT stores?

Quoted message said:

Quoted message said:

> > No. The IR arises as a consequence of VAT and not SAT.

Quoted message said:

Quoted message said:

> Can it be possible that IR starts on start of VAT formation, when SAT
> stores are exausted and increases on increase in VAT?

Quoted message said:

Quoted message said:

IR starts because of harmful inflammatory cytokines from VAT.

Quoted message said:

Start of of harmful inflammatory cytokines starts with start of VAT
forming and start of VAT forming will be on exhausting of SAT sores?

VAT starts forming when there is overeating.

Whether SAT and VAT formations can go together or VAT follow after
SAT formations are exhausted?

Quoted message said:

Quoted message said:

Quoted message said:

> It looks, VAT are formed may be for ugent/immediate energy need, if
> SAT's breakdown somehow are resisted to be used probably due to more
> and continual insulin?

Quoted message said:

Quoted message said:

Incorrect.

Quoted message said:

Though it is unclear but immediate enegy availability from VAT is
indicative in some states. Probably, VAT formation may occur in cases
of breakdown of enegy stores are resisted?

VAT formation occurs when there is overeating.

Logically, it looks bit unclear that self body's oriented creations
i.e. VAT formation has no purpose?
in contributing to some useful purpose of survival. Why not SAT go on
increasing instead VAT formation? Vascular densities in VAT may be
more, accordingly-- it may serve a purpose of immediate energy store
though some harmful.

Quoted message said:

Quoted message said:

Quoted message said:

> Though VAT may be considered pathological but
> still may have some purpose?

Quoted message said:

Quoted message said:

The purpose is to cause pathology... to cause harm.

Whether VATformation is a body' oriented mechanism or not?

Quoted message said:

Quoted message said:

Can't there be some pathology but more benefit for survival--immediate
available of energy on need if other energy stores are restricted/
resisted?

Such is the purpose of glycogen and not VAT.

I think glycogen's breakdown is also effected due to more and
contimual action of insulin whereas insulin don't effect VAT's
breakdown?

" Few Insulin's actions:
Increased glycogen synthesis - insulin forces storage of glucose in
liver (and muscle) cells in the form of glycogen; lowered levels of
insulin cause liver cells to convert glycogen to glucose and excrete
it into the blood. This is the clinical action of insulin which is
directly useful in reducing high blood glucose levels as in diabetes.
Increased esterification of fatty acids - forces adipose tissue to
make fats (ie, triglycerides) from fatty acid esters; lack of insulin
causes the reverse.
Decreased lipolysis - forces reduction in conversion of fat cell lipid
stores into blood fatty acids; lack of insulin causes the reverse.
Decreased gluconeogenesis - decreases production of glucose from
various substrates in liver; lack of insulin causes glucose production
from assorted substrates in the liver and elsewhere.
http://en.wikipedia.org/wiki/Insulin "

In absence of or if breakdown of energy stores are restricted due to
more and continual action of insulin, how body can derive energy for
its immediate need?

Quoted message said:

Quoted message said:

Quoted message said:

May GOD bless you.

Quoted message said:

Thanks.

You are welcome.

Have redirected your thanks to GOD so we will both be blessed.

Prayerfully in Jesus' ever-lasting love,

Andrew <><
--
Andrew B. Chung, MD/PhDhttp://EmoryCardiology.com

May HIS immortal brethren pray for our dying mortal friends and
neighbors:http://HeartMDPhD.com/Convicts

Especially dear Bob(this one) Pastorio:http://bobs-amanuensis.livejournal.com/4211.htmlhttp://pics.livejournal.com/bobs_amanuensis/pic/0000z24f/g1- Hide quoted text -

- Show quoted text -

convicted neighbor Kumar said:

Andrew said:

convicted neighbor Kumar said:

Andrew, in the Holy Spirit, boldly wrote:
> neighbor Kumar wrote:
> > Andrew, in the Holy Spirit, boldly wrote:
> > > neighbor Kumar wrote:
> > > > Andrew, in the Holy Spirit, boldly wrote:

Quoted message said:

> > snip> > > > Whether VATs are formed independetly of total fats status
> > or above
> > > > > > SAT?

Quoted message said:

> > > > > In excess of SAT stores.

Quoted message said:

> > > > In pre-VAT formation state on exhausting SAT stores capacity, will
> > > > there be some kind of resistance or IR to furthur SAT stores?

Quoted message said:

> > > No. The IR arises as a consequence of VAT and not SAT.

Quoted message said:

> > Can it be possible that IR starts on start of VAT formation, when SAT
> > stores are exausted and increases on increase in VAT?

Quoted message said:

> IR starts because of harmful inflammatory cytokines from VAT.

Quoted message said:

Start of of harmful inflammatory cytokines starts with start of VAT
forming and start of VAT forming will be on exhausting of SAT sores?

VAT starts forming when there is overeating.

Whether SAT and VAT formations can go together or VAT follow after
SAT formations are exhausted?

Depends on GOD's design of your body.

Wiser to not overeat.

Quoted message said:

Quoted message said:

Quoted message said:

> > It looks, VAT are formed may be for ugent/immediate energy need, if
> > SAT's breakdown somehow are resisted to be used probably due to more
> > and continual insulin?

Quoted message said:

> Incorrect.

Quoted message said:

Though it is unclear but immediate enegy availability from VAT is
indicative in some states. Probably, VAT formation may occur in cases
of breakdown of enegy stores are resisted?

VAT formation occurs when there is overeating.

Logically, it looks bit unclear that self body's oriented creations
i.e. VAT formation has no purpose?

VAT formation is the penalty for the sin of overeating (gluttony).

Quoted message said:

in contributing to some useful purpose of survival. Why not SAT go on
increasing instead VAT formation? Vascular densities in VAT may be
more, accordingly-- it may serve a purpose of immediate energy store
though some harmful.

"The wages of sin is death." -- Holy Spirit

Amen.

Quoted message said:

Quoted message said:

Quoted message said:

> > Though VAT may be considered pathological but
> > still may have some purpose?

Quoted message said:

> The purpose is to cause pathology... to cause harm.

Whether VATformation is a body' oriented mechanism or not?

It is a GOD mandated mechanism of chastening.

Quoted message said:

Quoted message said:

Quoted message said:

Can't there be some pathology but more benefit for survival--immediate
available of energy on need if other energy stores are restricted/
resisted?

Such is the purpose of glycogen and not VAT.

I think glycogen's breakdown is also effected due to more and
contimual action of insulin whereas insulin don't effect VAT's
breakdown?

Glycogenolysis is actually dependent on less insulin and not more.

Quoted message said:

" Few Insulin's actions:
Increased glycogen synthesis - insulin forces storage of glucose in
liver (and muscle) cells in the form of glycogen; lowered levels of
insulin cause liver cells to convert glycogen to glucose and excrete
it into the blood. This is the clinical action of insulin which is
directly useful in reducing high blood glucose levels as in diabetes.
Increased esterification of fatty acids - forces adipose tissue to
make fats (ie, triglycerides) from fatty acid esters; lack of insulin
causes the reverse.
Decreased lipolysis - forces reduction in conversion of fat cell lipid
stores into blood fatty acids; lack of insulin causes the reverse.
Decreased gluconeogenesis - decreases production of glucose from
various substrates in liver; lack of insulin causes glucose production
from assorted substrates in the liver and elsewhere.
http://en.wikipedia.org/wiki/Insulin "

In absence of or if breakdown of energy stores are restricted due to
more and continual action of insulin, how body can derive energy for
its immediate need?

If the body has an immediate need for energy, glucose levels will be
lower thereby suppressing insulin release.

Andrew B. Chung, MD/PhD
http://EmoryCardiology.com

On Apr 1, 5:36 am, "Andrew B. Chung, MD/PhD" <[email hidden]>

Quoted message said:

convicted neighbor Kumar said:

Andrew said:

convicted neighbor Kumar wrote:
> Andrew, in the Holy Spirit, boldly wrote:
> > neighbor Kumar wrote:
> > > Andrew, in the Holy Spirit, boldly wrote:
> > > > neighbor Kumar wrote:
> > > > > Andrew, in the Holy Spirit, boldly wrote:

Quoted message said:

Quoted message said:

> > > snip> > > > Whether VATs are formed independetly of total fats status
> > > or above
> > > > > > > SAT?

Quoted message said:

Quoted message said:

> > > > > > In excess of SAT stores.

Quoted message said:

Quoted message said:

> > > > > In pre-VAT formation state on exhausting SAT stores capacity, will
> > > > > there be some kind of resistance or IR to furthur SAT stores?

Quoted message said:

Quoted message said:

> > > > No. The IR arises as a consequence of VAT and not SAT.

Quoted message said:

Quoted message said:

> > > Can it be possible that IR starts on start of VAT formation, when SAT
> > > stores are exausted and increases on increase in VAT?

Quoted message said:

Quoted message said:

> > IR starts because of harmful inflammatory cytokines from VAT.

Quoted message said:

Quoted message said:

> Start of of harmful inflammatory cytokines starts with start of VAT
> forming and start of VAT forming will be on exhausting of SAT sores?

Quoted message said:

Quoted message said:

VAT starts forming when there is overeating.

Quoted message said:

Whether SAT and VAT formations can go together or VAT follow after
SAT formations are exhausted?

Depends on GOD's design of your body.

Pls tell me about few GOD's designs in this respect?

Quoted message said:

Wiser to not overeat.

Yes, that is absolute unless there is some medical reason.

Quoted message said:

Quoted message said:

Quoted message said:

> > > It looks, VAT are formed may be for ugent/immediate energy need, if
> > > SAT's breakdown somehow are resisted to be used probably due to more
> > > and continual insulin?

Quoted message said:

Quoted message said:

> > Incorrect.

Quoted message said:

Quoted message said:

> Though it is unclear but immediate enegy availability from VAT is
> indicative in some states. Probably, VAT formation may occur in cases
> of breakdown of enegy stores are resisted?

Quoted message said:

Quoted message said:

VAT formation occurs when there is overeating.

Quoted message said:

Logically, it looks bit unclear that self body's oriented creations
i.e. VAT formation has no purpose?

VAT formation is the penalty for the sin of overeating (gluttony).

Can't it be a penalty for some other disorder--natural or manmade?

Quoted message said:

Quoted message said:

in contributing to some useful purpose of survival. Why not SAT go on
increasing instead VAT formation? Vascular densities in VAT may be
more, accordingly-- it may serve a purpose of immediate energy store
though some harmful.

"The wages of sin is death." -- Holy Spirit

Amen.

Quoted message said:

Quoted message said:

> > > Though VAT may be considered pathological but
> > > still may have some purpose?

Quoted message said:

Quoted message said:

> > The purpose is to cause pathology... to cause harm.

Quoted message said:

Whether VATformation is a body' oriented mechanism or not?

It is a GOD mandated mechanism of chastening.

Quoted message said:

Quoted message said:

> Can't there be some pathology but more benefit for survival--immediate
> available of energy on need if other energy stores are restricted/
> resisted?

Quoted message said:

Quoted message said:

Such is the purpose of glycogen and not VAT.

Quoted message said:

I think glycogen's breakdown is also effected due to more and
contimual action of insulin whereas insulin don't effect VAT's
breakdown?

Glycogenolysis is actually dependent on less insulin and not more.

Yes I meant glycogen's breakdown effected, means lesser
Glycogenolysis due to more and continual insulin's exposure as per
indicated action of insulin. As such, if more and continual insulin's
exposure can reduce Glycogenolysis, then, immediate energy may not be
available from this source. However, if VAT's breakdown is non-insulin
dependant then energy can be derived from this(VAT) source/stores?

Quoted message said:

Quoted message said:

" Few Insulin's actions:
Increased glycogen synthesis - insulin forces storage of glucose in
liver (and muscle) cells in the form of glycogen; lowered levels of
insulin cause liver cells to convert glycogen to glucose and excrete
it into the blood. This is the clinical action of insulin which is
directly useful in reducing high blood glucose levels as in diabetes.
Increased esterification of fatty acids - forces adipose tissue to
make fats (ie, triglycerides) from fatty acid esters; lack of insulin
causes the reverse.
Decreased lipolysis - forces reduction in conversion of fat cell lipid
stores into blood fatty acids; lack of insulin causes the reverse.
Decreased gluconeogenesis - decreases production of glucose from
various substrates in liver; lack of insulin causes glucose production
from assorted substrates in the liver and elsewhere.
en.wikipedia.orgInsulin"Open ↗

Quoted message said:

In absence of or if breakdown of energy stores are restricted due to
more and continual action of insulin, how body can derive energy for
its immediate need?

If the body has an immediate need for energy, glucose levels will be
lower thereby suppressing insulin release.

What about on exposure of exogenous insulin esp. long acting?

Moreover this action may vary in insulin resistant cases?

On breakdown of VAT, whether body gets some immediate energy or not?

Whether VAT have more blood circulations than SAT?

Quoted message said:

Andrew B. Chung, MD/PhDhttp://EmoryCardiology.com- Hide quoted text -

- Show quoted text -- Hide quoted text -

- Show quoted text -

convicted neighbor Kumar said:

Andrew said:

convicted neighbor Kumar said:

Andrew, in the Holy Spirit, boldly wrote:
> convicted neighbor Kumar wrote:
> > Andrew, in the Holy Spirit, boldly wrote:
> > > neighbor Kumar wrote:
> > > > Andrew, in the Holy Spirit, boldly wrote:
> > > > > neighbor Kumar wrote:
> > > > > > Andrew, in the Holy Spirit, boldly wrote:

Quoted message said:

> > > > snip> > > > Whether VATs are formed independetly of total fats status
> > > > or above
> > > > > > > > SAT?

Quoted message said:

> > > > > > > In excess of SAT stores.

Quoted message said:

> > > > > > In pre-VAT formation state on exhausting SAT stores capacity, will
> > > > > > there be some kind of resistance or IR to furthur SAT stores?

Quoted message said:

> > > > > No. The IR arises as a consequence of VAT and not SAT.

Quoted message said:

> > > > Can it be possible that IR starts on start of VAT formation, when SAT
> > > > stores are exausted and increases on increase in VAT?

Quoted message said:

> > > IR starts because of harmful inflammatory cytokines from VAT.

Quoted message said:

> > Start of of harmful inflammatory cytokines starts with start of VAT
> > forming and start of VAT forming will be on exhausting of SAT sores?

Quoted message said:

> VAT starts forming when there is overeating.

Quoted message said:

Whether SAT and VAT formations can go together or VAT follow after
SAT formations are exhausted?

Depends on GOD's design of your body.

Pls tell me about few GOD's designs in this respect?

Only GOD knows.

Quoted message said:

Quoted message said:

Wiser to not overeat.

Yes, that is absolute unless there is some medical reason.

There is no medical reason to overeat.

Quoted message said:

Quoted message said:

Quoted message said:

> > > > It looks, VAT are formed may be for ugent/immediate energy need, if
> > > > SAT's breakdown somehow are resisted to be used probably due to more
> > > > and continual insulin?

Quoted message said:

> > > Incorrect.

Quoted message said:

> > Though it is unclear but immediate enegy availability from VAT is
> > indicative in some states. Probably, VAT formation may occur in cases
> > of breakdown of enegy stores are resisted?

Quoted message said:

> VAT formation occurs when there is overeating.

Quoted message said:

Logically, it looks bit unclear that self body's oriented creations
i.e. VAT formation has no purpose?

VAT formation is the penalty for the sin of overeating (gluttony).

Can't it be a penalty for some other disorder--natural or manmade?

No.

Quoted message said:

Quoted message said:

Quoted message said:

in contributing to some useful purpose of survival. Why not SAT go on
increasing instead VAT formation? Vascular densities in VAT may be
more, accordingly-- it may serve a purpose of immediate energy store
though some harmful.

"The wages of sin is death." -- Holy Spirit

Amen.

Quoted message said:

> > > > Though VAT may be considered pathological but
> > > > still may have some purpose?

Quoted message said:

> > > The purpose is to cause pathology... to cause harm.

Quoted message said:

Whether VATformation is a body' oriented mechanism or not?

It is a GOD mandated mechanism of chastening.

Quoted message said:

> > Can't there be some pathology but more benefit for survival--immediate
> > available of energy on need if other energy stores are restricted/
> > resisted?

Quoted message said:

> Such is the purpose of glycogen and not VAT.

Quoted message said:

I think glycogen's breakdown is also effected due to more and
contimual action of insulin whereas insulin don't effect VAT's
breakdown?

Glycogenolysis is actually dependent on less insulin and not more.

Yes I meant glycogen's breakdown effected, means lesser
Glycogenolysis due to more and continual insulin's exposure as per
indicated action of insulin. As such, if more and continual insulin's
exposure can reduce Glycogenolysis, then, immediate energy may not be
from this source. However, if VAT's breakdown is non-insulin
dependant then energy can be derived from this(VAT) source/stores?

Again, if there is a need for energy, insulin levels will be lowered.

Quoted message said:

Quoted message said:

Quoted message said:

" Few Insulin's actions:
Increased glycogen synthesis - insulin forces storage of glucose in
liver (and muscle) cells in the form of glycogen; lowered levels of
insulin cause liver cells to convert glycogen to glucose and excrete
it into the blood. This is the clinical action of insulin which is
directly useful in reducing high blood glucose levels as in diabetes.
Increased esterification of fatty acids - forces adipose tissue to
make fats (ie, triglycerides) from fatty acid esters; lack of insulin
causes the reverse.
Decreased lipolysis - forces reduction in conversion of fat cell lipid
stores into blood fatty acids; lack of insulin causes the reverse.
Decreased gluconeogenesis - decreases production of glucose from
various substrates in liver; lack of insulin causes glucose production
from assorted substrates in the liver and elsewhere.
en.wikipedia.orgInsulin"Open ↗

Quoted message said:

In absence of or if breakdown of energy stores are restricted due to
more and continual action of insulin, how body can derive energy for
its immediate need?

If the body has an immediate need for energy, glucose levels will be
lower thereby suppressing insulin release.

What about on exposure of exogenous insulin esp. long acting?

Then there will be a higher risk of hypoglycemia.

Quoted message said:

Moreover this action may vary in insulin resistant cases?

The risk would remain.

Quoted message said:

On breakdown of VAT, whether body gets some immediate energy or not?

Nothing clinically significant.

Quoted message said:

Whether VAT have more blood circulations than SAT?

It is more vascular because of location.

May GOD bless you.

Prayerfully in Jesus' ever-lasting love,

Andrew <><
--
Andrew B. Chung, MD/PhD
http://EmoryCardiology.com

May HIS immortal brethren pray for our dying mortal friends and
neighbors:
http://HeartMDPhD.com/Convicts

Especially dear Bob(this one) Pastorio:
http://bobs-amanuensis.livejournal.com/4211.html
http://pics.livejournal.com/bobs_amanuensis/pic/0000z24f/g1

On Apr 1, 10:12 pm, "Andrew B. Chung, MD/PhD" <[email hidden]>

Quoted message said:

Quoted message said:

Quoted message said:

> > > > > snip> > > > Whether VATs are formed independetly of total fats status
> > > > > or above
> > > > > > > > > SAT?

Quoted message said:

Quoted message said:

> > > > > > > > In excess of SAT stores.

Quoted message said:

Quoted message said:

> > > > > > > In pre-VAT formation state on exhausting SAT stores capacity, will
> > > > > > > there be some kind of resistance or IR to furthur SAT stores?

Quoted message said:

Quoted message said:

> > > > > > No. The IR arises as a consequence of VAT and not SAT.

Quoted message said:

Quoted message said:

> > > > > Can it be possible that IR starts on start of VAT formation, when SAT
> > > > > stores are exausted and increases on increase in VAT?

Quoted message said:

Quoted message said:

> > > > IR starts because of harmful inflammatory cytokines from VAT.

Quoted message said:

Quoted message said:

> > > Start of of harmful inflammatory cytokines starts with start of VAT
> > > forming and start of VAT forming will be on exhausting of SAT sores?

Quoted message said:

Quoted message said:

> > VAT starts forming when there is overeating.

Quoted message said:

Quoted message said:

> Whether SAT and VAT formations can go together or VAT follow after
> SAT formations are exhausted?

Quoted message said:

Quoted message said:

Depends on GOD's design of your body.

Quoted message said:

Pls tell me about few GOD's designs in this respect?

Only GOD knows.

Then, how can we say for sure that, SAT and VAT formations depend on
GOD's designs ?

Quoted message said:

Quoted message said:

Quoted message said:

Wiser to not overeat.

Quoted message said:

Yes, that is absolute unless there is some medical reason.

There is no medical reason to overeat.

More and continual insulin. either endo or exogenous?

Quoted message said:

Quoted message said:

Quoted message said:

> > > > > It looks, VAT are formed may be for ugent/immediate energy need, if
> > > > > SAT's breakdown somehow are resisted to be used probably due to more
> > > > > and continual insulin?

Quoted message said:

Quoted message said:

> > > > Incorrect.

Quoted message said:

Quoted message said:

> > > Though it is unclear but immediate enegy availability from VAT is
> > > indicative in some states. Probably, VAT formation may occur in cases
> > > of breakdown of enegy stores are resisted?

Quoted message said:

Quoted message said:

> > VAT formation occurs when there is overeating.

Quoted message said:

Quoted message said:

> Logically, it looks bit unclear that self body's oriented creations
> i.e. VAT formation has no purpose?

Quoted message said:

Quoted message said:

VAT formation is the penalty for the sin of overeating (gluttony).

Quoted message said:

Can't it be a penalty for some other disorder--natural or manmade?

No.

Quoted message said:

Quoted message said:

> in contributing to some useful purpose of survival. Why not SAT go on
> increasing instead VAT formation? Vascular densities in VAT may be
> more, accordingly-- it may serve a purpose of immediate energy store
> though some harmful.

Quoted message said:

Quoted message said:

"The wages of sin is death." -- Holy Spirit

Quoted message said:

Quoted message said:

Amen.

Quoted message said:

Quoted message said:

> > > > > Though VAT may be considered pathological but
> > > > > still may have some purpose?

Quoted message said:

Quoted message said:

> > > > The purpose is to cause pathology... to cause harm.

Quoted message said:

Quoted message said:

> Whether VATformation is a body' oriented mechanism or not?

Quoted message said:

Quoted message said:

It is a GOD mandated mechanism of chastening.

Quoted message said:

Quoted message said:

> > > Can't there be some pathology but more benefit for survival--immediate
> > > available of energy on need if other energy stores are restricted/
> > > resisted?

Quoted message said:

Quoted message said:

> > Such is the purpose of glycogen and not VAT.

Quoted message said:

Quoted message said:

> I think glycogen's breakdown is also effected due to more and
> contimual action of insulin whereas insulin don't effect VAT's
> breakdown?

Quoted message said:

Quoted message said:

Glycogenolysis is actually dependent on less insulin and not more.

Quoted message said:

Yes I meant glycogen's breakdown effected, means lesser
Glycogenolysis due to more and continual insulin's exposure as per
indicated action of insulin. As such, if more and continual insulin's
exposure can reduce Glycogenolysis, then, immediate energy may not be
from this source. However, if VAT's breakdown is non-insulin
dependant then energy can be derived from this(VAT) source/stores?

Again, if there is a need for energy, insulin levels will be lowered.

It looks bit logical that, on exposure of more and continual insulin,
natural(prediabetic/IR case)
or exogenous, resist and restricts energy release from energy stores
which persisting hyperglycemia and/or VAT may compensate? May be,
these factors result into overeating. As such, why can't more and
continual exposure of insulin be the basic reason to getting diabetes2
related problems. In IDDM
there may not be exposure of more and continual insulin, so may get
better control from less insulin
?

Quoted message said:

Quoted message said:

Quoted message said:

> " Few Insulin's actions:
> Increased glycogen synthesis - insulin forces storage of glucose in
> liver (and muscle) cells in the form of glycogen; lowered levels of
> insulin cause liver cells to convert glycogen to glucose and excrete
> it into the blood. This is the clinical action of insulin which is
> directly useful in reducing high blood glucose levels as in diabetes.
> Increased esterification of fatty acids - forces adipose tissue to
> make fats (ie, triglycerides) from fatty acid esters; lack of insulin
> causes the reverse.
> Decreased lipolysis - forces reduction in conversion of fat cell lipid
> stores into blood fatty acids; lack of insulin causes the reverse.
> Decreased gluconeogenesis - decreases production of glucose from
> various substrates in liver; lack of insulin causes glucose production
> from assorted substrates in the liver and elsewhere.
>http://en.wikipedia.org/wiki/Insulin"

Quoted message said:

Quoted message said:

> In absence of or if breakdown of energy stores are restricted due to
> more and continual action of insulin, how body can derive energy for
> its immediate need?

Quoted message said:

Quoted message said:

If the body has an immediate need for energy, glucose levels will be
lower thereby suppressing insulin release.

Quoted message said:

What about on exposure of exogenous insulin esp. long acting?

Then there will be a higher risk of hypoglycemia.

In case of insulin resistant people?

Quoted message said:

Quoted message said:

Moreover this action may vary in insulin resistant cases?

The risk would remain.

May be, therefore a need to maintain energy from persisting
hyperglycemia, IR and VAT's stores?

Quoted message said:

Quoted message said:

On breakdown of VAT, whether body gets some immediate energy or not?

Nothing clinically significant.

But the amount of VATs in diabetics can be significant? As such, Why
its breakdown can't provide significant and immediate energy(due to
more vascular as you said)?

Quoted message said:

Quoted message said:

Whether VAT have more blood circulations than SAT?

Quoted message said:

It is more vascular because of location.

May GOD bless you.

HE MAY as my intentions remain true and pure which may make me getting
HIS graces and resistent to many adversities?

Quoted message said:

Prayerfully in Jesus' ever-lasting love,

Andrew <><
--
Andrew B. Chung, MD/PhDhttp://EmoryCardiology.com

May HIS immortal brethren pray for our dying mortal friends and
neighbors:http://HeartMDPhD.com/Convicts

Especially dear Bob(this one) Pastorio:http://bobs-amanuensis.livejournal.com/4211.htmlhttp://pics.livejournal.com/bobs_amanuensis/pic/0000z24f/g1- Hide quoted text -

- Show quoted text -- Hide quoted text -

- Show quoted text -- Hide quoted text -

- Show quoted text -- Hide quoted text -

- Show quoted text -

convicted neighbor Kumar said:

Andrew said:

Quoted message said:

> > > > > > snip> > > > Whether VATs are formed independetly of total fats status
> > > > > > or above
> > > > > > > > > > SAT?

Quoted message said:

> > > > > > > > > In excess of SAT stores.

Quoted message said:

> > > > > > > > In pre-VAT formation state on exhausting SAT stores capacity, will
> > > > > > > > there be some kind of resistance or IR to furthur SAT stores?

Quoted message said:

> > > > > > > No. The IR arises as a consequence of VAT and not SAT.

Quoted message said:

> > > > > > Can it be possible that IR starts on start of VAT formation, when SAT
> > > > > > stores are exausted and increases on increase in VAT?

Quoted message said:

> > > > > IR starts because of harmful inflammatory cytokines from VAT.

Quoted message said:

> > > > Start of of harmful inflammatory cytokines starts with start of VAT
> > > > forming and start of VAT forming will be on exhausting of SAT sores?

Quoted message said:

> > > VAT starts forming when there is overeating.

Quoted message said:

> > Whether SAT and VAT formations can go together or VAT follow after
> > SAT formations are exhausted?

Quoted message said:

> Depends on GOD's design of your body.

Quoted message said:

Pls tell me about few GOD's designs in this respect?

Only GOD knows.

Then, how can we say for sure that, SAT and VAT formations depend on
GOD's designs ?

HE created us.

LORD Almighty GOD is the Creator of the universe and Author of all
reality (even the alternate ones).

Quoted message said:

Quoted message said:

Quoted message said:

> Wiser to not overeat.

Quoted message said:

Yes, that is absolute unless there is some medical reason.

There is no medical reason to overeat.

More and continual insulin. either endo or exogenous?

No.

Quoted message said:

Quoted message said:

Quoted message said:

> > > > > > It looks, VAT are formed may be for ugent/immediate energy need, if
> > > > > > SAT's breakdown somehow are resisted to be used probably due to more
> > > > > > and continual insulin?

Quoted message said:

> > > > > Incorrect.

Quoted message said:

> > > > Though it is unclear but immediate enegy availability from VAT is
> > > > indicative in some states. Probably, VAT formation may occur in cases
> > > > of breakdown of enegy stores are resisted?

Quoted message said:

> > > VAT formation occurs when there is overeating.

Quoted message said:

> > Logically, it looks bit unclear that self body's oriented creations
> > i.e. VAT formation has no purpose?

Quoted message said:

> VAT formation is the penalty for the sin of overeating (gluttony).

Quoted message said:

Can't it be a penalty for some other disorder--natural or manmade?

No.

Quoted message said:

> > in contributing to some useful purpose of survival. Why not SAT go on
> > increasing instead VAT formation? Vascular densities in VAT may be
> > more, accordingly-- it may serve a purpose of immediate energy store
> > though some harmful.

Quoted message said:

> "The wages of sin is death." -- Holy Spirit

Quoted message said:

> Amen.

Quoted message said:

> > > > > > Though VAT may be considered pathological but
> > > > > > still may have some purpose?

Quoted message said:

> > > > > The purpose is to cause pathology... to cause harm.

Quoted message said:

> > Whether VATformation is a body' oriented mechanism or not?

Quoted message said:

> It is a GOD mandated mechanism of chastening.

Quoted message said:

> > > > Can't there be some pathology but more benefit for survival--immediate
> > > > available of energy on need if other energy stores are restricted/
> > > > resisted?

Quoted message said:

> > > Such is the purpose of glycogen and not VAT.

Quoted message said:

> > I think glycogen's breakdown is also effected due to more and
> > contimual action of insulin whereas insulin don't effect VAT's
> > breakdown?

Quoted message said:

> Glycogenolysis is actually dependent on less insulin and not more.

Quoted message said:

Yes I meant glycogen's breakdown effected, means lesser
Glycogenolysis due to more and continual insulin's exposure as per
indicated action of insulin. As such, if more and continual insulin's
exposure can reduce Glycogenolysis, then, immediate energy may not be
from this source. However, if VAT's breakdown is non-insulin
dependant then energy can be derived from this(VAT) source/stores?

Again, if there is a need for energy, insulin levels will be lowered.

It looks bit logical that, on exposure of more and continual insulin,
natural(prediabetic/IR case)
or exogenous, resist and restricts energy release from energy stores
which persisting hyperglycemia and/or VAT may compensate?

VAT is pathological.

Quoted message said:

May be,
these factors result into overeating. As such, why can't more and
continual exposure of insulin be the basic reason to getting diabetes2

No.

Quoted message said:

related problems. In IDDM
there may not be exposure of more and continual insulin, so may get
better control from less insulin ?

IDDM do not typically have better control compared to Type-2
diabetics.

Quoted message said:

Quoted message said:

Quoted message said:

> > " Few Insulin's actions:
> > Increased glycogen synthesis - insulin forces storage of glucose in
> > liver (and muscle) cells in the form of glycogen; lowered levels of
> > insulin cause liver cells to convert glycogen to glucose and excrete
> > it into the blood. This is the clinical action of insulin which is
> > directly useful in reducing high blood glucose levels as in diabetes.
> > Increased esterification of fatty acids - forces adipose tissue to
> > make fats (ie, triglycerides) from fatty acid esters; lack of insulin
> > causes the reverse.
> > Decreased lipolysis - forces reduction in conversion of fat cell lipid
> > stores into blood fatty acids; lack of insulin causes the reverse.
> > Decreased gluconeogenesis - decreases production of glucose from
> > various substrates in liver; lack of insulin causes glucose production
> > from assorted substrates in the liver and elsewhere.
> >http://en.wikipedia.org/wiki/Insulin"

Quoted message said:

> > In absence of or if breakdown of energy stores are restricted due to
> > more and continual action of insulin, how body can derive energy for
> > its immediate need?

Quoted message said:

> If the body has an immediate need for energy, glucose levels will be
> lower thereby suppressing insulin release.

Quoted message said:

What about on exposure of exogenous insulin esp. long acting?

Then there will be a higher risk of hypoglycemia.

In case of insulin resistant people?

Correct.

Quoted message said:

Quoted message said:

Quoted message said:

Moreover this action may vary in insulin resistant cases?

The risk would remain.

May be, therefore a need to maintain energy from persisting
hyperglycemia, IR and VAT's stores?

No.

Quoted message said:

Quoted message said:

Quoted message said:

On breakdown of VAT, whether body gets some immediate energy or not?

Nothing clinically significant.

But the amount of VATs in diabetics can be significant? As such, Why
its breakdown can't provide significant and immediate energy(due to
more vascular as you said)?

By GOD's design.

Quoted message said:

Quoted message said:

Quoted message said:

Whether VAT have more blood circulations than SAT?

Quoted message said:

It is more vascular because of location.

May GOD bless you.

HE MAY as my intentions remain true and pure which may make me getting
HIS graces and resistent to many adversities?

GOD's will be done and not our wills.

Andrew B. Chung, MD/PhD
http://EmoryCardiology.com

On Apr 2, 6:27 pm, "Andrew B. Chung, MD/PhD"
<[email hidden]> wrote:
snip> > > Again, if there is a need for energy, insulin levels will be
lowered.

Quoted message said:

Quoted message said:

It looks bit logical that, on exposure of more and continual insulin,
natural(prediabetic/IR case)
or exogenous, resist and restricts energy release from energy stores
which persisting hyperglycemia and/or VAT may compensate?

VAT is pathological.

Do we get many conditions in our body which are some pathological but
beneficial in nett for survival?

Quoted message said:

Quoted message said:

May be,
these factors result into overeating. As such, why can't more and
continual exposure of insulin be the basic reason to getting diabetes2

No.

Quoted message said:

related problems. In IDDM
there may not be exposure of more and continual insulin, so may get
better control from less insulin ?

IDDM do not typically have better control compared to Type-2
diabetics.

How insulin's effects are different in IDDM and in NIDDM?

Quoted message said:

Quoted message said:

Quoted message said:

> > > " Few Insulin's actions:
> > > Increased glycogen synthesis - insulin forces storage of glucose in
> > > liver (and muscle) cells in the form of glycogen; lowered levels of
> > > insulin cause liver cells to convert glycogen to glucose and excrete
> > > it into the blood. This is the clinical action of insulin which is
> > > directly useful in reducing high blood glucose levels as in diabetes.
> > > Increased esterification of fatty acids - forces adipose tissue to
> > > make fats (ie, triglycerides) from fatty acid esters; lack of insulin
> > > causes the reverse.
> > > Decreased lipolysis - forces reduction in conversion of fat cell lipid
> > > stores into blood fatty acids; lack of insulin causes the reverse.
> > > Decreased gluconeogenesis - decreases production of glucose from
> > > various substrates in liver; lack of insulin causes glucose production
> > > from assorted substrates in the liver and elsewhere.
> > >http://en.wikipedia.org/wiki/Insulin"

Quoted message said:

Quoted message said:

> > > In absence of or if breakdown of energy stores are restricted due to
> > > more and continual action of insulin, how body can derive energy for
> > > its immediate need?

Quoted message said:

Quoted message said:

> > If the body has an immediate need for energy, glucose levels will be
> > lower thereby suppressing insulin release.

Quoted message said:

Quoted message said:

> What about on exposure of exogenous insulin esp. long acting?

Quoted message said:

Quoted message said:

Then there will be a higher risk of hypoglycemia.

Quoted message said:

In case of insulin resistant people?

Correct.

Btw, insulin is less effective in IR cases and so glucose uptake is
impaired. As such, why a diabetic2 with IR don't get hypoglycemic type
symtoms?

Quoted message said:

Quoted message said:

Quoted message said:

> Moreover this action may vary in insulin resistant cases?

Quoted message said:

Quoted message said:

The risk would remain.

Quoted message said:

May be, therefore a need to maintain energy from persisting
hyperglycemia, IR and VAT's stores?

No.

Sorry but, due to indicated actions of insulin about decreased
breakdown of energy stores, on more and continual exposure of insulin
above thought can persist.

Quoted message said:

Quoted message said:

Quoted message said:

> On breakdown of VAT, whether body gets some immediate energy or not?

Quoted message said:

Quoted message said:

Nothing clinically significant.

Quoted message said:

But the amount of VATs in diabetics can be significant? As such, Why
its breakdown can't provide significant and immediate energy(due to
more vascular as you said)?

By GOD's design.

How much energy we can get from VATs breakdown?

Quoted message said:

Quoted message said:

Quoted message said:

> Whether VAT have more blood circulations than SAT?

Quoted message said:

Quoted message said:

It is more vascular because of location.

Quoted message said:

Quoted message said:

May GOD bless you.

Quoted message said:

HE MAY as my intentions remain true and pure which may make me getting
HIS graces and resistent to many adversities?

GOD's will be done and not our wills.

We can only do our duty, rest HE will only decide.

Quoted message said:

Andrew B. Chung, MD/PhDhttp://EmoryCardiology.com- Hide quoted text -

- Show quoted text -- Hide quoted text -

- Show quoted text -- Hide quoted text -

- Show quoted text -

convicted neighbor Kumar said:

Andrew, in the Holy Spirit, boldly wrote:

snip> > > Again, if there is a need for energy, insulin levels will be
lowered.

Quoted message said:

Quoted message said:

It looks bit logical that, on exposure of more and continual insulin,
natural(prediabetic/IR case)
or exogenous, resist and restricts energy release from energy stores
which persisting hyperglycemia and/or VAT may compensate?

VAT is pathological.

Do we get many conditions in our body which are some pathological but
beneficial in nett for survival?

There is nothing beneficial about VAT.

Quoted message said:

Quoted message said:

Quoted message said:

May be,
these factors result into overeating. As such, why can't more and
continual exposure of insulin be the basic reason to getting diabetes2

No.

Quoted message said:

related problems. In IDDM
there may not be exposure of more and continual insulin, so may get
better control from less insulin ?

IDDM do not typically have better control compared to Type-2
diabetics.

How insulin's effects are different in IDDM and in NIDDM?

No difference in overall insulin effect in type-1 compared to type-2
diabetics except in the size of the effect.

Quoted message said:

Quoted message said:

Quoted message said:

> > > > " Few Insulin's actions:
> > > > Increased glycogen synthesis - insulin forces storage of glucose in
> > > > liver (and muscle) cells in the form of glycogen; lowered levels of
> > > > insulin cause liver cells to convert glycogen to glucose and excrete
> > > > it into the blood. This is the clinical action of insulin which is
> > > > directly useful in reducing high blood glucose levels as in diabetes.
> > > > Increased esterification of fatty acids - forces adipose tissue to
> > > > make fats (ie, triglycerides) from fatty acid esters; lack of insulin
> > > > causes the reverse.
> > > > Decreased lipolysis - forces reduction in conversion of fat cell lipid
> > > > stores into blood fatty acids; lack of insulin causes the reverse.
> > > > Decreased gluconeogenesis - decreases production of glucose from
> > > > various substrates in liver; lack of insulin causes glucose production
> > > > from assorted substrates in the liver and elsewhere.
> > > >http://en.wikipedia.org/wiki/Insulin"

Quoted message said:

> > > > In absence of or if breakdown of energy stores are restricted due to
> > > > more and continual action of insulin, how body can derive energy for
> > > > its immediate need?

Quoted message said:

> > > If the body has an immediate need for energy, glucose levels will be
> > > lower thereby suppressing insulin release.

Quoted message said:

> > What about on exposure of exogenous insulin esp. long acting?

Quoted message said:

> Then there will be a higher risk of hypoglycemia.

Quoted message said:

In case of insulin resistant people?

Correct.

Btw, insulin is less effective in IR cases and so glucose uptake is
impaired. As such, why a diabetic2 with IR don't get hypoglycemic type
symtoms?

They do when they become hypoglycemic.

Quoted message said:

Quoted message said:

Quoted message said:

> > Moreover this action may vary in insulin resistant cases?

Quoted message said:

> The risk would remain.

Quoted message said:

May be, therefore a need to maintain energy from persisting
hyperglycemia, IR and VAT's stores?

No.

Sorry but, due to indicated actions of insulin about decreased
breakdown of energy stores, on more and continual exposure of insulin
above thought can persist.

The problem arises from overeating and not from insulin exposure.

Quoted message said:

Quoted message said:

Quoted message said:

> > On breakdown of VAT, whether body gets some immediate energy or not?

Quoted message said:

> Nothing clinically significant.

Quoted message said:

But the amount of VATs in diabetics can be significant? As such, Why
its breakdown can't provide significant and immediate energy(due to
more vascular as you said)?

By GOD's design.

How much energy we can get from VATs breakdown?

Nothing that we actually need because there are glycogen and SAT
stores which are non-pathological.

Quoted message said:

Quoted message said:

Quoted message said:

> > Whether VAT have more blood circulations than SAT?

Quoted message said:

> It is more vascular because of location.

Quoted message said:

> May GOD bless you.

Quoted message said:

HE MAY as my intentions remain true and pure which may make me getting
HIS graces and resistent to many adversities?

GOD's will be done and not our wills.

We can only do our duty, rest HE will only decide.

Our duty is to seek HIM out, know HIS commandments, and then obey HIM.

May GOD bless you in HIS mighty way.

Prayerfully in Jesus' ever-lasting love,

Andrew <><
--
Andrew B. Chung, MD/PhD
http://EmoryCardiology.com

May HIS immortal brethren pray for our dying mortal friends and
neighbors:
http://HeartMDPhD.com/Convicts

In memory of our dearly departed Bob(this one) Pastorio:
http://HeartMDPhD.com/Convicts/Bob

As for knowing who are the very elect, these you will know by the
unconditional love they have for everyone including their enemies
(Matthew 5:44-45, 1 Corinthians 13:3, James 2:14-17).
http://HeartMDPhD.com/Love

About the 2PD-OMER Approach:
http://abchung.livejournal.com/986.html?thread=16090#t16090

The Official SMC FAQ List:
http://tinyurl.com/3bshdc

Andrew B. Chung said:

convicted neighbor Kumar said:

Andrew, in the Holy Spirit, boldly wrote:

snip> > > Again, if there is a need for energy, insulin levels will be
lowered.

Quoted message said:

> It looks bit logical that, on exposure of more and continual insulin,
> natural(prediabetic/IR case)
> or exogenous, resist and restricts energy release from energy stores
> which persisting hyperglycemia and/or VAT may compensate?

VAT is pathological.

Do we get many conditions in our body which are some pathological but
beneficial in nett for survival?

There is nothing beneficial about VAT.

Quoted message said:

Quoted message said:

> May be,
> these factors result into overeating. As such, why can't more and
> continual exposure of insulin be the basic reason to getting diabetes2

No.

> related problems. In IDDM
> there may not be exposure of more and continual insulin, so may get
> better control from less insulin ?

IDDM do not typically have better control compared to Type-2
diabetics.

How insulin's effects are different in IDDM and in NIDDM?

No difference in overall insulin effect in type-1 compared to type-2
diabetics except in the size of the effect.

Quoted message said:

Quoted message said:

> > > > > " Few Insulin's actions:
> > > > > Increased glycogen synthesis - insulin forces storage of glucose in
> > > > > liver (and muscle) cells in the form of glycogen; lowered levels of
> > > > > insulin cause liver cells to convert glycogen to glucose and excrete
> > > > > it into the blood. This is the clinical action of insulin which is
> > > > > directly useful in reducing high blood glucose levels as in diabetes.
> > > > > Increased esterification of fatty acids - forces adipose tissue to
> > > > > make fats (ie, triglycerides) from fatty acid esters; lack of insulin
> > > > > causes the reverse.
> > > > > Decreased lipolysis - forces reduction in conversion of fat cell lipid
> > > > > stores into blood fatty acids; lack of insulin causes the reverse.
> > > > > Decreased gluconeogenesis - decreases production of glucose from
> > > > > various substrates in liver; lack of insulin causes glucose production
> > > > > from assorted substrates in the liver and elsewhere.
> > > > >http://en.wikipedia.org/wiki/Insulin"

> > > > > In absence of or if breakdown of energy stores are restricted due to
> > > > > more and continual action of insulin, how body can derive energy for
> > > > > its immediate need?

> > > > If the body has an immediate need for energy, glucose levels will be
> > > > lower thereby suppressing insulin release.

> > > What about on exposure of exogenous insulin esp. long acting?

> > Then there will be a higher risk of hypoglycemia.

> In case of insulin resistant people?

Correct.

Btw, insulin is less effective in IR cases and so glucose uptake is
impaired. As such, why a diabetic2 with IR don't get hypoglycemic type
symtoms?

They do when they become hypoglycemic.

Whether hypoglycemia just result into lesser availability so lesser
uptake of glucose by target cells or also causes some other
instabilities? Insulin resistance also express similar condition i.e.
lesser uptake by target cells?

Quoted message said:

Quoted message said:

Quoted message said:

> > > Moreover this action may vary in insulin resistant cases?

> > The risk would remain.

> May be, therefore a need to maintain energy from persisting
> hyperglycemia, IR and VAT's stores?

No.

Sorry but, due to indicated actions of insulin about decreased
breakdown of energy stores, on more and continual exposure of insulin
above thought can persist.

The problem arises from overeating and not from insulin exposure.

Overeatings and exogenous insulin can cause more and continual
insulin's exposures.

Quoted message said:

Quoted message said:

Quoted message said:

> > > On breakdown of VAT, whether body gets some immediate energy or not?

> > Nothing clinically significant.

> But the amount of VATs in diabetics can be significant? As such, Why
> its breakdown can't provide significant and immediate energy(due to
> more vascular as you said)?

By GOD's design.

How much energy we can get from VATs breakdown?

Nothing that we actually need because there are glycogen and SAT
stores which are non-pathological.

But energy from these stores can be resisted or restricted due to more
and continual exposure of exo/endo genous insulin. Then in such case,
how body will get immediate needed energy?

Quoted message said:

Quoted message said:

Quoted message said:

> > > Whether VAT have more blood circulations than SAT?

> > It is more vascular because of location.

> > May GOD bless you.

> HE MAY as my intentions remain true and pure which may make me getting
> HIS graces and resistent to many adversities?

GOD's will be done and not our wills.

We can only do our duty, rest HE will only decide.

Our duty is to seek HIM out, know HIS commandments, and then obey HIM.

Pls tell me more about " seek HIM out".

Quoted message said:

May GOD bless you in HIS mighty way.

Prayerfully in Jesus' ever-lasting love,

Andrew <><
--
Andrew B. Chung, MD/PhD
http://EmoryCardiology.com

May HIS immortal brethren pray for our dying mortal friends and
neighbors:
http://HeartMDPhD.com/Convicts

In memory of our dearly departed Bob(this one) Pastorio:
http://HeartMDPhD.com/Convicts/Bob

As for knowing who are the very elect, these you will know by the
unconditional love they have for everyone including their enemies
(Matthew 5:44-45, 1 Corinthians 13:3, James 2:14-17).
http://HeartMDPhD.com/Love

About the 2PD-OMER Approach:
http://abchung.livejournal.com/986.html?thread=16090#t16090

The Official SMC FAQ List:
http://tinyurl.com/3bshdc

convicted neighbor Kumar said:

Andrew, in the Holy Spirit, boldlywrote:

Quoted message said:

convicted neighbor Kumar said:

Andrew, in the Holy Spirit, boldly wrote:

snip> > > Again, if there is a need for energy, insulin levels will be
lowered.
>
> > It looks bit logical that, on exposure of more and continual insulin,
> > natural(prediabetic/IR case)
> > or exogenous, resist and restricts energy release from energy stores
> > which persisting hyperglycemia and/or VAT may compensate?
>
> VAT is pathological.

Do we get many conditions in our body which are some pathological but
beneficial in nett for survival?

There is nothing beneficial about VAT.

Quoted message said:

> > May be,
> > these factors result into overeating. As such, why can't more and
> > continual exposure of insulin be the basic reason to getting diabetes2
>
> No.
>
> > related problems. In IDDM
> > there may not be exposure of more and continual insulin, so may get
> > better control from less insulin ?
>
> IDDM do not typically have better control compared to Type-2
> diabetics.

How insulin's effects are different in IDDM and in NIDDM?

No difference in overall insulin effect in type-1 compared to type-2
diabetics except in the size of the effect.

Quoted message said:

> > > > > > " Few Insulin's actions:
> > > > > > Increased glycogen synthesis - insulin forces storage of glucose in
> > > > > > liver (and muscle) cells in the form of glycogen; lowered levels of
> > > > > > insulin cause liver cells to convert glycogen to glucose and excrete
> > > > > > it into the blood. This is the clinical action of insulin which is
> > > > > > directly useful in reducing high blood glucose levels as in diabetes.
> > > > > > Increased esterification of fatty acids - forces adipose tissue to
> > > > > > make fats (ie, triglycerides) from fatty acid esters; lack of insulin
> > > > > > causes the reverse.
> > > > > > Decreased lipolysis - forces reduction in conversion of fat cell lipid
> > > > > > stores into blood fatty acids; lack of insulin causes the reverse.
> > > > > > Decreased gluconeogenesis - decreases production of glucose from
> > > > > > various substrates in liver; lack of insulin causes glucose production
> > > > > > from assorted substrates in the liver and elsewhere.
> > > > > >http://en.wikipedia.org/wiki/Insulin"
>
> > > > > > In absence of or if breakdown of energy stores are restricted due to
> > > > > > more and continual action of insulin, how body can derive energy for
> > > > > > its immediate need?
>
> > > > > If the body has an immediate need for energy, glucose levels will be
> > > > > lower thereby suppressing insulin release.
>
> > > > What about on exposure of exogenous insulin esp. long acting?
>
> > > Then there will be a higher risk of hypoglycemia.
>
> > In case of insulin resistant people?
>
> Correct.

Btw, insulin is less effective in IR cases and so glucose uptake is
impaired. As such, why a diabetic2 with IR don't get hypoglycemic type
symtoms?

They do when they become hypoglycemic.

Whether hypoglycemia just result into lesser availability so lesser
uptake of glucose by target cells or also causes some other
instabilities? Insulin resistance also express similar condition i.e.
lesser uptake by target cells?

Insulin resistance and hypoglycemia are not synonymous.

Quoted message said:

Quoted message said:

Quoted message said:

> > > > Moreover this action may vary in insulin resistant cases?
>
> > > The risk would remain.
>
> > May be, therefore a need to maintain energy from persisting
> > hyperglycemia, IR and VAT's stores?
>
> No.

Sorry but, due to indicated actions of insulin about decreased
breakdown of energy stores, on more and continual exposure of insulin
above thought can persist.

The problem arises from overeating and not from insulin exposure.

Overeatings and exogenous insulin can cause more and continual
insulin's exposures.

Stop overeating and the problem is solved.

Quoted message said:

Quoted message said:

Quoted message said:

> > > > On breakdown of VAT, whether body gets some immediate energy or not?
>
> > > Nothing clinically significant.
>
> > But the amount of VATs in diabetics can be significant? As such, Why
> > its breakdown can't provide significant and immediate energy(due to
> > more vascular as you said)?
>
> By GOD's design.

How much energy we can get from VATs breakdown?

Nothing that we actually need because there are glycogen and SAT
stores which are non-pathological.

But energy from these stores can be resisted or restricted due to more
and continual exposure of exo/endo genous insulin. Then in such case,
how body will get immediate needed energy?

Either for glycogenolysis or gluconeogenesis.

Quoted message said:

Quoted message said:

Quoted message said:

> > > > Whether VAT have more blood circulations than SAT?
>
> > > It is more vascular because of location.
>
> > > May GOD bless you.
>
> > HE MAY as my intentions remain true and pure which may make me getting
> > HIS graces and resistent to many adversities?
>
> GOD's will be done and not our wills.

We can only do our duty, rest HE will only decide.

Our duty is to seek HIM out, know HIS commandments, and then obey HIM.

Pls tell me more about " seek HIM out".

Prayer.

May GOD bless you.

Prayerfully in Jesus' ever-lasting love,

Andrew <><
--
Andrew B. Chung, MD/PhD
http://EmoryCardiology.com

May HIS immortal brethren pray for our dying mortal friends and
neighbors:
http://HeartMDPhD.com/Convicts

In memory of our dearly departed Bob(this one) Pastorio:
http://HeartMDPhD.com/Convicts/Bob

As for knowing who are the very elect, these you will know by the
unconditional love they have for everyone including their enemies
(Matthew 5:44-45, 1 Corinthians 13:3, James 2:14-17).
http://HeartMDPhD.com/Love

MD/PhD said:

convicted neighbor Kumar said:

Andrew, in the Holy Spirit, boldlywrote:

Quoted message said:

convicted neighbor Kumar wrote:
> Andrew, in the Holy Spirit, boldly wrote:

Quoted message said:

Quoted message said:

> snip> > > Again, if there is a need for energy, insulin levels will be
> lowered.

Quoted message said:

Quoted message said:

> > > It looks bit logical that, on exposure of more and continual insulin,
> > > natural(prediabetic/IR case)
> > > or exogenous, resist and restricts energy release from energy stores
> > > which persisting hyperglycemia and/or VAT may compensate?

Quoted message said:

Quoted message said:

> > VAT is pathological.

Quoted message said:

Quoted message said:

> Do we get many conditions in our body which are some pathological but
> beneficial in nett for survival?

Quoted message said:

Quoted message said:

There is nothing beneficial about VAT.

Quoted message said:

Quoted message said:

> > > May be,
> > > these factors result into overeating. As such, why can't more and
> > > continual exposure of insulin be the basic reason to getting diabetes2

Quoted message said:

Quoted message said:

> > No.

Quoted message said:

Quoted message said:

> > > related problems. In IDDM
> > > there may not be exposure of more and continual insulin, so may get
> > > better control from less insulin ?

Quoted message said:

Quoted message said:

> > IDDM do not typically have better control compared to Type-2
> > diabetics.

Quoted message said:

Quoted message said:

> How insulin's effects are different in IDDM and in NIDDM?

Quoted message said:

Quoted message said:

No difference in overall insulin effect in type-1 compared to type-2
diabetics except in the size of the effect.

Quoted message said:

Quoted message said:

> > > > > > > " Few Insulin's actions:
> > > > > > > Increased glycogen synthesis - insulin forces storage of glucose in
> > > > > > > liver (and muscle) cells in the form of glycogen; lowered levels of
> > > > > > > insulin cause liver cells to convert glycogen to glucose and excrete
> > > > > > > it into the blood. This is the clinical action of insulin which is
> > > > > > > directly useful in reducing high blood glucose levels as in diabetes.
> > > > > > > Increased esterification of fatty acids - forces adipose tissue to
> > > > > > > make fats (ie, triglycerides) from fatty acid esters; lack of insulin
> > > > > > > causes the reverse.
> > > > > > > Decreased lipolysis - forces reduction in conversion of fat cell lipid
> > > > > > > stores into blood fatty acids; lack of insulin causes the reverse.
> > > > > > > Decreased gluconeogenesis - decreases production of glucose from
> > > > > > > various substrates in liver; lack of insulin causes glucose production
> > > > > > > from assorted substrates in the liver and elsewhere.
> > > > > > >http://en.wikipedia.org/wiki/Insulin"

Quoted message said:

Quoted message said:

> > > > > > > In absence of or if breakdown of energy stores are restricted due to
> > > > > > > more and continual action of insulin, how body can derive energy for
> > > > > > > its immediate need?

Quoted message said:

Quoted message said:

> > > > > > If the body has an immediate need for energy, glucose levels will be
> > > > > > lower thereby suppressing insulin release.

Quoted message said:

Quoted message said:

> > > > > What about on exposure of exogenous insulin esp. long acting?

Quoted message said:

Quoted message said:

> > > > Then there will be a higher risk of hypoglycemia.

Quoted message said:

Quoted message said:

> > > In case of insulin resistant people?

Quoted message said:

Quoted message said:

> > Correct.

Quoted message said:

Quoted message said:

> Btw, insulin is less effective in IR cases and so glucose uptake is
> impaired. As such, why a diabetic2 with IR don't get hypoglycemic type
> symtoms?

Quoted message said:

Quoted message said:

They do when they become hypoglycemic.

Quoted message said:

Whether hypoglycemia just result into lesser availability so lesser
uptake of glucose by target cells or also causes some other
instabilities? Insulin resistance also express similar condition i.e.
lesser uptake by target cells?

Insulin resistance and hypoglycemia are not synonymous.

Yes that can be a thought to understand what really a so thought
hypoglycemia is, either lack of glucose or an instability of minerals
or other body's constituents caused by either low glucose or by low
insulin?

Quoted message said:

Quoted message said:

Quoted message said:

> > > > > Moreover this action may vary in insulin resistant cases?

Quoted message said:

Quoted message said:

> > > > The risk would remain.

Quoted message said:

Quoted message said:

> > > May be, therefore a need to maintain energy from persisting
> > > hyperglycemia, IR and VAT's stores?

Quoted message said:

Quoted message said:

> > No.

Quoted message said:

Quoted message said:

> Sorry but, due to indicated actions of insulin about decreased
> breakdown of energy stores, on more and continual exposure of insulin
> above thought can persist.

Quoted message said:

Quoted message said:

The problem arises from overeating and not from insulin exposure.

Quoted message said:

Overeatings and exogenous insulin can cause more and continual
insulin's exposures.

Stop overeating and the problem is solved.

Yes but that can be the main cause of getting the disease. No meaning
of medical involment in many cases, if a patient can overcome the main
cause. But the problem can be in overcoming the main cause.

Quoted message said:

Quoted message said:

Quoted message said:

> > > > > On breakdown of VAT, whether body gets some immediate energy or not?

Quoted message said:

Quoted message said:

> > > > Nothing clinically significant.

Quoted message said:

Quoted message said:

> > > But the amount of VATs in diabetics can be significant? As such, Why
> > > its breakdown can't provide significant and immediate energy(due to
> > > more vascular as you said)?

Quoted message said:

Quoted message said:

> > By GOD's design.

Quoted message said:

Quoted message said:

> How much energy we can get from VATs breakdown?

Quoted message said:

Quoted message said:

Nothing that we actually need because there are glycogen and SAT
stores which are non-pathological.

Quoted message said:

But energy from these stores can be resisted or restricted due to more
and continual exposure of exo/endo genous insulin. Then in such case,
how body will get immediate needed energy?

Either for glycogenolysis or gluconeogenesis.

How on more and continual exposure of exo/endo genous insulin which
may resist energy stores breakdown?

Alike other energy stores, can more and continual insulin's exposure
also resist or restrict breakdown of VATs?

Quoted message said:

Quoted message said:

Quoted message said:

> > > > > Whether VAT have more blood circulations than SAT?

Quoted message said:

Quoted message said:

> > > > It is more vascular because of location.

Quoted message said:

Quoted message said:

> > > > May GOD bless you.

Quoted message said:

Quoted message said:

> > > HE MAY as my intentions remain true and pure which may make me getting
> > > HIS graces and resistent to many adversities?

Quoted message said:

Quoted message said:

> > GOD's will be done and not our wills.

Quoted message said:

Quoted message said:

> We can only do our duty, rest HE will only decide.

Quoted message said:

Quoted message said:

Our duty is to seek HIM out, know HIS commandments, and then obey HIM.

Quoted message said:

Pls tell me more about " seek HIM out".

Prayer.

What do you mean by " seek HIM out"?

Quoted message said:

May GOD bless you.

Prayerfully in Jesus' ever-lasting love,

Andrew <><
--
Andrew B. Chung, MD/PhDhttp://EmoryCardiology.com

May HIS immortal brethren pray for our dying mortal friends and
neighbors:http://HeartMDPhD.com/Convicts

In memory of our dearly departed Bob(this one) Pastorio:http://HeartMDPhD.com/Convicts/Bob

As for knowing who are the very elect, these you will know by the
unconditional love they have for everyone including their enemies
(Matthew 5:44-45, 1 Corinthians 13:3, James 2:14-17).http://HeartMDPhD.com/Love- Hide quoted text -

- Show quoted text -- Hide quoted text -

- Show quoted text -- Hide quoted text -

- Show quoted text -- Hide quoted text -

- Show quoted text -

convicted neighbor Kumar said:

Andrew said:

convicted neighbor Kumar said:

Andrew, in the Holy Spirit, boldlywrote:
> convicted neighbor Kumar wrote:
> > Andrew, in the Holy Spirit, boldly wrote:

Quoted message said:

> > snip> > > Again, if there is a need for energy, insulin levels will be
> > lowered.

Quoted message said:

> > > > It looks bit logical that, on exposure of more and continual insulin,
> > > > natural(prediabetic/IR case)
> > > > or exogenous, resist and restricts energy release from energy stores
> > > > which persisting hyperglycemia and/or VAT may compensate?

Quoted message said:

> > > VAT is pathological.

Quoted message said:

> > Do we get many conditions in our body which are some pathological but
> > beneficial in nett for survival?

Quoted message said:

> There is nothing beneficial about VAT.

Quoted message said:

> > > > May be,
> > > > these factors result into overeating. As such, why can't more and
> > > > continual exposure of insulin be the basic reason to getting diabetes2

Quoted message said:

> > > No.

Quoted message said:

> > > > related problems. In IDDM
> > > > there may not be exposure of more and continual insulin, so may get
> > > > better control from less insulin ?

Quoted message said:

> > > IDDM do not typically have better control compared to Type-2
> > > diabetics.

Quoted message said:

> > How insulin's effects are different in IDDM and in NIDDM?

Quoted message said:

> No difference in overall insulin effect in type-1 compared to type-2
> diabetics except in the size of the effect.

Quoted message said:

> > > > > > > > " Few Insulin's actions:
> > > > > > > > Increased glycogen synthesis - insulin forces storage of glucose in
> > > > > > > > liver (and muscle) cells in the form of glycogen; lowered levels of
> > > > > > > > insulin cause liver cells to convert glycogen to glucose and excrete
> > > > > > > > it into the blood. This is the clinical action of insulin which is
> > > > > > > > directly useful in reducing high blood glucose levels as in diabetes.
> > > > > > > > Increased esterification of fatty acids - forces adipose tissue to
> > > > > > > > make fats (ie, triglycerides) from fatty acid esters; lack of insulin
> > > > > > > > causes the reverse.
> > > > > > > > Decreased lipolysis - forces reduction in conversion of fat cell lipid
> > > > > > > > stores into blood fatty acids; lack of insulin causes the reverse.
> > > > > > > > Decreased gluconeogenesis - decreases production of glucose from
> > > > > > > > various substrates in liver; lack of insulin causes glucose production
> > > > > > > > from assorted substrates in the liver and elsewhere.
> > > > > > > >http://en.wikipedia.org/wiki/Insulin"

Quoted message said:

> > > > > > > > In absence of or if breakdown of energy stores are restricted due to
> > > > > > > > more and continual action of insulin, how body can derive energy for
> > > > > > > > its immediate need?

Quoted message said:

> > > > > > > If the body has an immediate need for energy, glucose levels will be
> > > > > > > lower thereby suppressing insulin release.

Quoted message said:

> > > > > > What about on exposure of exogenous insulin esp. long acting?

Quoted message said:

> > > > > Then there will be a higher risk of hypoglycemia.

Quoted message said:

> > > > In case of insulin resistant people?

Quoted message said:

> > > Correct.

Quoted message said:

> > Btw, insulin is less effective in IR cases and so glucose uptake is
> > impaired. As such, why a diabetic2 with IR don't get hypoglycemic type
> > symtoms?

Quoted message said:

> They do when they become hypoglycemic.

Quoted message said:

Whether hypoglycemia just result into lesser availability so lesser
uptake of glucose by target cells or also causes some other
instabilities? Insulin resistance also express similar condition i.e.
lesser uptake by target cells?

Insulin resistance and hypoglycemia are not synonymous.

Yes that can be a thought to understand what really a so thought
hypoglycemia is, either lack of glucose or an instability of minerals
or other body's constituents caused by either low glucose or by low
insulin?

Hypoglycemia is simply low serum glucose and not instability of
minerals.

Quoted message said:

Quoted message said:

Quoted message said:

> > > > > > Moreover this action may vary in insulin resistant cases?

Quoted message said:

> > > > > The risk would remain.

Quoted message said:

> > > > May be, therefore a need to maintain energy from persisting
> > > > hyperglycemia, IR and VAT's stores?

Quoted message said:

> > > No.

Quoted message said:

> > Sorry but, due to indicated actions of insulin about decreased
> > breakdown of energy stores, on more and continual exposure of insulin
> > above thought can persist.

Quoted message said:

> The problem arises from overeating and not from insulin exposure.

Quoted message said:

Overeatings and exogenous insulin can cause more and continual
insulin's exposures.

Stop overeating and the problem is solved.

Yes but that can be the main cause of getting the disease.

Not can but **is** the cause of the disease.

Quoted message said:

No meaning
of medical involment in many cases, if a patient can overcome the main
cause. But the problem can be in overcoming the main cause.

Simply stop overeating:

http://HeartMDPhD.com/HolySpirit/overweight.asp

Works every time. There is now even a one million dollar guarantee:

http://TruthRUS.org/Guarantee

Suggested reading:

http://abchung.livejournal.com/986.html?thread=16090#t16090

Quoted message said:

Quoted message said:

Quoted message said:

> > > > > > On breakdown of VAT, whether body gets some immediate energy or not?

Quoted message said:

> > > > > Nothing clinically significant.

Quoted message said:

> > > > But the amount of VATs in diabetics can be significant? As such, Why
> > > > its breakdown can't provide significant and immediate energy(due to
> > > > more vascular as you said)?

Quoted message said:

> > > By GOD's design.

Quoted message said:

> > How much energy we can get from VATs breakdown?

Quoted message said:

> Nothing that we actually need because there are glycogen and SAT
> stores which are non-pathological.

Quoted message said:

But energy from these stores can be resisted or restricted due to more
and continual exposure of exo/endo genous insulin. Then in such case,
how body will get immediate needed energy?

Either by glycogenolysis or gluconeogenesis.

How on more and continual exposure of exo/endo genous insulin which
may resist energy stores breakdown?

Insulin levels will be low when there is need for energy.

Quoted message said:

Alike other energy stores, can more and continual insulin's exposure
also resist or restrict breakdown of VATs?

Insulin levels will be high when there is no need for energy.

Quoted message said:

Quoted message said:

Quoted message said:

> > > > > > Whether VAT have more blood circulations than SAT?

Quoted message said:

> > > > > It is more vascular because of location.

Quoted message said:

> > > > > May GOD bless you.

Quoted message said:

> > > > HE MAY as my intentions remain true and pure which may make me getting
> > > > HIS graces and resistent to many adversities?

Quoted message said:

> > > GOD's will be done and not our wills.

Quoted message said:

> > We can only do our duty, rest HE will only decide.

Quoted message said:

> Our duty is to seek HIM out, know HIS commandments, and then obey HIM.

Quoted message said:

Pls tell me more about " seek HIM out".

Prayer.

Quoted message said:

What do you mean by " seek HIM out"?

http://abchung.livejournal.com/1176.html?thread=21144#t21144

May GOD bless you.

Prayerfully in Jesus' ever-lasting love,

Andrew <><
--
Andrew B. Chung, MD/PhD
http://EmoryCardiology.com

May HIS immortal brethren pray for our dying mortal friends and
neighbors:
http://HeartMDPhD.com/Convicts

In memory of our dearly departed Bob(this one) Pastorio:
http://HeartMDPhD.com/Convicts/Bob

As for knowing who are the very elect, these you will know by the
unconditional love they have for everyone including their enemies
(Matthew 5:44-45, 1 Corinthians 13:3, James 2:14-17).
http://HeartMDPhD.com/Love

Andrew B. Chung said:

convicted neighbor Kumar said:

Andrew said:

convicted neighbor Kumar wrote:
> Andrew, in the Holy Spirit, boldlywrote:
> > convicted neighbor Kumar wrote:
> > > Andrew, in the Holy Spirit, boldly wrote:

> > > snip> > > Again, if there is a need for energy, insulin levels will be
> > > lowered.

> > > > > It looks bit logical that, on exposure of more and continual insulin,
> > > > > natural(prediabetic/IR case)
> > > > > or exogenous, resist and restricts energy release from energy stores
> > > > > which persisting hyperglycemia and/or VAT may compensate?

> > > > VAT is pathological.

> > > Do we get many conditions in our body which are some pathological but
> > > beneficial in nett for survival?

> > There is nothing beneficial about VAT.

> > > > > May be,
> > > > > these factors result into overeating. As such, why can't more and
> > > > > continual exposure of insulin be the basic reason to getting diabetes2

> > > > No.

> > > > > related problems. In IDDM
> > > > > there may not be exposure of more and continual insulin, so may get
> > > > > better control from less insulin ?

> > > > IDDM do not typically have better control compared to Type-2
> > > > diabetics.

> > > How insulin's effects are different in IDDM and in NIDDM?

> > No difference in overall insulin effect in type-1 compared to type-2
> > diabetics except in the size of the effect.

> > > > > > > > > " Few Insulin's actions:
> > > > > > > > > Increased glycogen synthesis - insulin forces storage of glucose in
> > > > > > > > > liver (and muscle) cells in the form of glycogen; lowered levels of
> > > > > > > > > insulin cause liver cells to convert glycogen to glucose and excrete
> > > > > > > > > it into the blood. This is the clinical action of insulin which is
> > > > > > > > > directly useful in reducing high blood glucose levels as in diabetes.
> > > > > > > > > Increased esterification of fatty acids - forces adipose tissue to
> > > > > > > > > make fats (ie, triglycerides) from fatty acid esters; lack of insulin
> > > > > > > > > causes the reverse.
> > > > > > > > > Decreased lipolysis - forces reduction in conversion of fat cell lipid
> > > > > > > > > stores into blood fatty acids; lack of insulin causes the reverse.
> > > > > > > > > Decreased gluconeogenesis - decreases production of glucose from
> > > > > > > > > various substrates in liver; lack of insulin causes glucose production
> > > > > > > > > from assorted substrates in the liver and elsewhere.
> > > > > > > > >http://en.wikipedia.org/wiki/Insulin"

> > > > > > > > > In absence of or if breakdown of energy stores are restricted due to
> > > > > > > > > more and continual action of insulin, how body can derive energy for
> > > > > > > > > its immediate need?

> > > > > > > > If the body has an immediate need for energy, glucose levels will be
> > > > > > > > lower thereby suppressing insulin release.

> > > > > > > What about on exposure of exogenous insulin esp. long acting?

> > > > > > Then there will be a higher risk of hypoglycemia.

> > > > > In case of insulin resistant people?

> > > > Correct.

> > > Btw, insulin is less effective in IR cases and so glucose uptake is
> > > impaired. As such, why a diabetic2 with IR don't get hypoglycemic type
> > > symtoms?

> > They do when they become hypoglycemic.

> Whether hypoglycemia just result into lesser availability so lesser
> uptake of glucose by target cells or also causes some other
> instabilities? Insulin resistance also express similar condition i.e.
> lesser uptake by target cells?

Insulin resistance and hypoglycemia are not synonymous.

Yes that can be a thought to understand what really a so thought
hypoglycemia is, either lack of glucose or an instability of minerals
or other body's constituents caused by either low glucose or by low
insulin?

Hypoglycemia is simply low serum glucose and not instability of
minerals.

In real, hypoglycemia symptoms are just due to low serum glucose or
lesser glucose exposure to target cells?
Thanks.

Quoted message said:

May GOD bless you.

Prayerfully in Jesus' ever-lasting love,

Andrew <><
--
Andrew B. Chung, MD/PhD
http://EmoryCardiology.com

May HIS immortal brethren pray for our dying mortal friends and
neighbors:
http://HeartMDPhD.com/Convicts

In memory of our dearly departed Bob(this one) Pastorio:
http://HeartMDPhD.com/Convicts/Bob

As for knowing who are the very elect, these you will know by the
unconditional love they have for everyone including their enemies
(Matthew 5:44-45, 1 Corinthians 13:3, James 2:14-17).
http://HeartMDPhD.com/Love

convicted neighbor Kumar said:

Andrew said:

convicted neighbor Kumar said:

Andrew, in the Holy Spirit, boldly wrote:
> convicted neighbor Kumar wrote:
> > Andrew, in the Holy Spirit, boldlywrote:
> > > convicted neighbor Kumar wrote:
> > > > Andrew, in the Holy Spirit, boldly wrote:
>
> > > > snip> > > Again, if there is a need for energy, insulin levels will be
> > > > lowered.
>
> > > > > > It looks bit logical that, on exposure of more and continual insulin,
> > > > > > natural(prediabetic/IR case)
> > > > > > or exogenous, resist and restricts energy release from energy stores
> > > > > > which persisting hyperglycemia and/or VAT may compensate?
>
> > > > > VAT is pathological.
>
> > > > Do we get many conditions in our body which are some pathological but
> > > > beneficial in nett for survival?
>
> > > There is nothing beneficial about VAT.
>
> > > > > > May be,
> > > > > > these factors result into overeating. As such, why can't more and
> > > > > > continual exposure of insulin be the basic reason to getting diabetes2
>
> > > > > No.
>
> > > > > > related problems. In IDDM
> > > > > > there may not be exposure of more and continual insulin, so may get
> > > > > > better control from less insulin ?
>
> > > > > IDDM do not typically have better control compared to Type-2
> > > > > diabetics.
>
> > > > How insulin's effects are different in IDDM and in NIDDM?
>
> > > No difference in overall insulin effect in type-1 compared to type-2
> > > diabetics except in the size of the effect.
>
> > > > > > > > > > " Few Insulin's actions:
> > > > > > > > > > Increased glycogen synthesis - insulin forces storage of glucose in
> > > > > > > > > > liver (and muscle) cells in the form of glycogen; lowered levels of
> > > > > > > > > > insulin cause liver cells to convert glycogen to glucose and excrete
> > > > > > > > > > it into the blood. This is the clinical action of insulin which is
> > > > > > > > > > directly useful in reducing high blood glucose levels as in diabetes.
> > > > > > > > > > Increased esterification of fatty acids - forces adipose tissue to
> > > > > > > > > > make fats (ie, triglycerides) from fatty acid esters; lack of insulin
> > > > > > > > > > causes the reverse.
> > > > > > > > > > Decreased lipolysis - forces reduction in conversion of fat cell lipid
> > > > > > > > > > stores into blood fatty acids; lack of insulin causes the reverse.
> > > > > > > > > > Decreased gluconeogenesis - decreases production of glucose from
> > > > > > > > > > various substrates in liver; lack of insulin causes glucose production
> > > > > > > > > > from assorted substrates in the liver and elsewhere.
> > > > > > > > > >http://en.wikipedia.org/wiki/Insulin"
>
> > > > > > > > > > In absence of or if breakdown of energy stores are restricted due to
> > > > > > > > > > more and continual action of insulin, how body can derive energy for
> > > > > > > > > > its immediate need?
>
> > > > > > > > > If the body has an immediate need for energy, glucose levels will be
> > > > > > > > > lower thereby suppressing insulin release.
>
> > > > > > > > What about on exposure of exogenous insulin esp. long acting?
>
> > > > > > > Then there will be a higher risk of hypoglycemia.
>
> > > > > > In case of insulin resistant people?
>
> > > > > Correct.
>
> > > > Btw, insulin is less effective in IR cases and so glucose uptake is
> > > > impaired. As such, why a diabetic2 with IR don't get hypoglycemic type
> > > > symtoms?
>
> > > They do when they become hypoglycemic.
>
> > Whether hypoglycemia just result into lesser availability so lesser
> > uptake of glucose by target cells or also causes some other
> > instabilities? Insulin resistance also express similar condition i.e.
> > lesser uptake by target cells?
>
> Insulin resistance and hypoglycemia are not synonymous.

Yes that can be a thought to understand what really a so thought
hypoglycemia is, either lack of glucose or an instability of minerals
or other body's constituents caused by either low glucose or by low
insulin?

Hypoglycemia is simply low serum glucose and not instability of
minerals.

In real, hypoglycemia symptoms are just due to low serum glucose or
lesser glucose exposure to target cells?

Low serum glucose.

Quoted message said:

Thanks.

You are welcome, dear Kumar.

Would redirect all thanks to GOD so that we will both be blessed.

Prayerfully in Jesus' ever-lasting love,

Andrew <><
--
Andrew B. Chung, MD/PhD
http://EmoryCardiology.com

May HIS immortal brethren pray for our dying mortal friends and
neighbors:
http://HeartMDPhD.com/Convicts

In memory of our dearly departed Bob(this one) Pastorio:
http://HeartMDPhD.com/Convicts/Bob

Andrew B. Chung said:

convicted neighbor Kumar said:

Andrew, in the Holy Spirit, boldly wrote:

snip> > > Hypoglycemia is simply low serum glucose and not instability
of

Quoted message said:

Quoted message said:

Quoted message said:

minerals.

In real, hypoglycemia symptoms are just due to low serum glucose or
lesser glucose exposure to target cells?

Low serum glucose.

Will low serum glucose and lesser exposure of glucose not occur same
time?

Quoted message said:

Quoted message said:

Thanks.

You are welcome, dear Kumar.

Would redirect all thanks to GOD so that we will both be blessed.

Prayerfully in Jesus' ever-lasting love,

Andrew <><
--
Andrew B. Chung, MD/PhD
http://EmoryCardiology.com

May HIS immortal brethren pray for our dying mortal friends and
neighbors:
http://HeartMDPhD.com/Convicts

In memory of our dearly departed Bob(this one) Pastorio:
http://HeartMDPhD.com/Convicts/Bob

convicted neighbor Kumar said:

Andrew said:

convicted neighbor Kumar said:

Andrew, in the Holy Spirit, boldly wrote:

snip> > > Hypoglycemia is simply low serum glucose and not instability
of

Quoted message said:

Quoted message said:

> minerals.

In real, hypoglycemia symptoms are just due to low serum glucose or
lesser glucose exposure to target cells?

Low serum glucose.

Will low serum glucose and lesser exposure of glucose not occur same
time?

What is measured is serum glucose.

May GOD bless you in HIS mighty way.

Prayerfully in Jesus' ever-lasting love,

Andrew <><
--
Andrew B. Chung, MD/PhD
http://EmoryCardiology.com

May HIS immortal brethren pray for our dying mortal friends and
neighbors:
http://HeartMDPhD.com/Convicts

In memory of our dearly departed Bob(this one) Pastorio:
http://HeartMDPhD.com/Convicts/Bob

As for knowing who are the very elect, these you will know by the
unconditional love they have for everyone including their enemies
(Matthew 5:44-45, 1 Corinthians 13:3, James 2:14-17).
http://HeartMDPhD.com/Love

The Official SMC FAQ List:
http://tinyurl.com/3bshdc

On Apr 8, 11:50 pm, "Andrew B. Chung, MD/PhD" <[email hidden]>

Quoted message said:

convicted neighbor Kumar said:

Andrew said:

convicted neighbor Kumar wrote:
> Andrew, in the Holy Spirit, boldly wrote:

Quoted message said:

snip> > > Hypoglycemia is simply low serum glucose and not instability
of

Quoted message said:

> > minerals.

Quoted message said:

Quoted message said:

> In real, hypoglycemia symptoms are just due to low serum glucose or
> lesser glucose exposure to target cells?

Quoted message said:

Quoted message said:

Low serum glucose.

Quoted message said:

Will low serum glucose and lesser exposure of glucose not occur same
time?

What is measured is serum glucose.

Yes. Thanks for your replies. Will participate actively bit later.

Quoted message said:

May GOD bless you in HIS mighty way.

Thanks for your prayers.

Quoted message said:

Prayerfully in Jesus' ever-lasting love,

Andrew <><
--
Andrew B. Chung, MD/PhDhttp://EmoryCardiology.com

May HIS immortal brethren pray for our dying mortal friends and
neighbors:http://HeartMDPhD.com/Convicts

In memory of our dearly departed Bob(this one) Pastorio:http://HeartMDPhD.com/Convicts/Bob

As for knowing who are the very elect, these you will know by the
unconditional love they have for everyone including their enemies
(Matthew 5:44-45, 1 Corinthians 13:3, James 2:14-17).http://HeartMDPhD.com/Love

The Official SMC FAQ List:http://tinyurl.com/3bshdc- Hide quoted text -

- Show quoted text -

convicted neighbor Kumar said:

Andrew said:

convicted neighbor Kumar said:

Andrew, in the Holy Spirit, boldly wrote:
> convicted neighbor Kumar wrote:
> > Andrew, in the Holy Spirit, boldly wrote:

Quoted message said:

snip> > > Hypoglycemia is simply low serum glucose and not instability
of
> > > minerals.

Quoted message said:

> > In real, hypoglycemia symptoms are just due to low serum glucose or
> > lesser glucose exposure to target cells?

Quoted message said:

> Low serum glucose.

Quoted message said:

Will low serum glucose and lesser exposure of glucose not occur same
time?

What is measured is serum glucose.

Yes. Thanks for your replies. Will participate actively bit later.

Quoted message said:

May GOD bless you in HIS mighty way.

Thanks for your prayers.

Thanks be to GOD.

Prayerfully in Jesus' ever-lasting love,

Andrew <><
--
Andrew B. Chung, MD/PhD
http://EmoryCardiology.com

May HIS immortal brethren pray for our dying mortal friends and
neighbors:
http://HeartMDPhD.com/Convicts

In memory of our dearly departed Bob(this one) Pastorio:
http://HeartMDPhD.com/Convicts/Bob

As for knowing who are the very elect, these you will know by the
unconditional love they have for everyone including their enemies
(Matthew 5:44-45, 1 Corinthians 13:3, James 2:14-17).
http://HeartMDPhD.com/Love

The Official SMC FAQ List:
http://HeartMDPhD.com/TheTruth/FAQ