Doesnt the notion that adopting a ketogenic diet instantly transports you into a permanent Zone 2 state oversimplify the intricacies of exercise physiology and metabolism, particularly considering the variability in individual responses to diet and exercise, the influence of factors such as fitness level, experience, and training specificity, as well as the unsubstantiated assumption that the body can adapt to maintain a state of elevated fat oxidation even in the face of intense exercise or prolonged periods of low-intensity activity?
How can proponents of this approach reconcile the lack of empirical evidence supporting the idea that a ketogenic diet induces a permanent shift in exercise intensity zones, especially when considering the dynamic interplay between factors like glycogen depletion, lactate threshold, and critical power? Dont these arguments for a permanent Zone 2 neglect the complex interplay between metabolic, cardiovascular, and neuromuscular factors that govern an athletes response to exercise, and cant they be debunked by the existence of numerous counterexamples where trained athletes on a ketogenic diet still exhibit significant variability in exercise intensity and perceived exertion?
Moreover, wouldnt it be inaccurate to imply that the benefits of a ketogenic diet are primarily derived from its effects on exercise intensity zones, when in fact the diets therapeutic applications span a far broader range of areas, including improved body composition, enhanced endurance capacity, and reduced systemic inflammation? Shouldnt advocates of ketogenic diets emphasize the need for a more nuanced understanding of their effects on exercise physiology, one that acknowledges the inherent variability and individuality of human metabolism, rather than relying on oversimplified and unsubstantiated claims about permanent Zone 2?
How can proponents of this approach reconcile the lack of empirical evidence supporting the idea that a ketogenic diet induces a permanent shift in exercise intensity zones, especially when considering the dynamic interplay between factors like glycogen depletion, lactate threshold, and critical power? Dont these arguments for a permanent Zone 2 neglect the complex interplay between metabolic, cardiovascular, and neuromuscular factors that govern an athletes response to exercise, and cant they be debunked by the existence of numerous counterexamples where trained athletes on a ketogenic diet still exhibit significant variability in exercise intensity and perceived exertion?
Moreover, wouldnt it be inaccurate to imply that the benefits of a ketogenic diet are primarily derived from its effects on exercise intensity zones, when in fact the diets therapeutic applications span a far broader range of areas, including improved body composition, enhanced endurance capacity, and reduced systemic inflammation? Shouldnt advocates of ketogenic diets emphasize the need for a more nuanced understanding of their effects on exercise physiology, one that acknowledges the inherent variability and individuality of human metabolism, rather than relying on oversimplified and unsubstantiated claims about permanent Zone 2?