Sat, 13 Dec 2003 23:22:52 GMT in article
<[email hidden]> "giselle" <[email hidden]>
Quoted message said:
"Matti Narkia" <[email hidden]> wrote.Quoted message said:Quoted message said:
The Cretan Mediterranean diet works best in Cretans who plow fields and walk 9 miles a day.
Is that so?Yes, it is so. Such were the Mediterraneans that Keys knew. In less active types, 40% MUFA is
likely to lead to excess fat in the body.
Not according to the following studies:
Willett WC, Leibel RL. Dietary fat is not a major determinant of body fat. Am J Med. 2002 Dec 30;113
Suppl 9B:47S-59S. Review. <ncbi.nlm.nih.govquery.fcgiOpen ↗
t_uids=12566139&dopt=Abstract> ( tinyurl.comwaizOpen ↗ )
"... in randomized trials lasting >or=1 year, fat consumption within the range of 18% to 40% of
energy appears to have little if any effect on body fatness. ..."
Willett WC. Dietary fat plays a major role in obesity: no. Obes Rev. 2002 May;3(2):59-68.
Review. <ncbi.nlm.nih.govquery.fcgiOpen ↗
1&dopt=Abstract>
"... Diets high in fat do not account for the high prevalence of excess body fat in Western
countries; reductions in the percentage of energy from fat will have no important benefits and
could further exacerbate this problem. The emphasis on total fat reduction has been a serious
distraction in efforts to control obesity and improve health in general."
Katan MB, Grundy SM, Willett WC. [Less fat or a different fat?] Ned Tijdschr Geneeskd. 1998 Apr 18;142(16):886-
9. Review. Dutch.
"Current dietary advice with a view to avoiding cardiovascular pathology is to replace fatty
foodstuffs and those rich in saturated fat and cholesterol by food rich in complex
carbohydrates. Although substitution of carbohydrates for fat lowers the blood level of low-
density lipoprotein
(LDL) cholesterol, it also lowers the level of high-density lipoprotein (HDL) cholesterol,
thereby adversely influencing the risk profile for cardiovascular disease. Neither does a
low fat diet appear to reduce obesity, another risk factor. A modern advice based on
published research reads: obese persons should reduce their intake of saturated and trans-
fatty acids by lowering the consumption of dairy fat, meat and hardened oils (bakery
products and catering products fried in hardened fats), and in addition should consume
less products with added sugars and refined starch. Carbohydrates should be provided by
fruits, vegetables, leguminous plants and whole-wheat products. Persons of about the ideal
weight should replace saturated and trans-fatty acids in their diet by unsaturated plant
oils, and products with refined carbohydrates by fruits, vegetables and whole-wheat
products."
Willett WC. Is dietary fat a major determinant of body fat? Am J Clin Nutr. 1998 Mar;67(3 Suppl):556S-
562S. Review. <ncbi.nlm.nih.govquery.fcgiOpen ↗
70&dopt=Abstract> ( tinyurl.comwamrOpen ↗ ) <http://www.ajcn.org/cgi/reprint/67/3/556S.pdf>
"The percentage of energy from dietary fat is widely believed to be an important determinant of
body fat, and several mechanisms have been proposed to account for such a relation. Comparisons
of both diets and the prevalence of obesity between affluent and poor countries have been used
to support a causal association, but these contrasts are seriously confounded by differences in
physical activity and food availability. Within areas of similar economic development, regional
intake of fat and prevalence of obesity have not been positively correlated. Randomized trials
are the preferable method to evaluate the effect of dietary fat on adiposity, and are feasible
because the number of subjects needed is not large. In short-term trials, a modest reduction in
body weight is typically seen in individuals randomly assigned to diets with a lower percentage
of energy from fat. However, compensatory mechanisms appear to operate because in trials lasting
Quoted message said:or = 1 y, fat consumption within the range of 18-40% of energy appears to have little if any
effect on body fatness. Moreover, within the United States, a substantial decline in the
percentage of energy from fat consumed during the past two decades has corresponded with a
massive increase in obesity. Diets high in fat do not appear to be the primary cause of the high
prevalence of excess body fat in our society, and reductions in fat will not be a solution."
Lichtenstein AH. Dietary fat and cardiovascular disease risk: quantity or quality? J Womens Health
(Larchmt). 2003 Mar;12(2):109-14. Review. <ncbi.nlm.nih.govquery.fcgiOpen ↗
ve&db=PubMed&list_uids=12737709&dopt=Abstract> ( tinyurl.comwaojOpen ↗ )
"When considering dietary fat quantity, there are two main factors to consider, impact on body
weight and plasma lipoprotein profiles. Data supporting a major role of dietary fat quantity in
determining body weight are weak and may be confounded by differences in energy density, dietary
fiber, and dietary protein. With respect to plasma lipoprotein profiles, relatively consistent
evidence indicates that under isoweight conditions, decreasing the total fat content of the diet
causes an increase in triglyceride and decrease in high- density lipoprotein (HDL) cholesterol
levels. When considering dietary fat quality, current evidence suggests that saturated fatty
acids tend to increase low-density lipoprotein (LDL) cholesterol levels, whereas monounsaturated
and polyunsaturated fatty acids tend to decrease LDL cholesterol levels. Long-chain omega-3
fatty acids, eicosapentaenoic acid (EPA) (20:5n-3) and docosahexaenoic acid (DHA) (22:6n-3), are
associated with decreased triglyceride levels in hypertriglyceridemic patients and decreased
risk of developing coronary heart disease (CHD). Dietary trans-fatty acids are associated with
increased LDL cholesterol levels. Hence, a diet low in saturated and trans- fatty acids, with
adequate amounts of monounsaturated and polyunsaturated fatty acids, especially long-chain omega-
3 fatty acids, would be recommended to reduce the risk of developing CHD. ..."
Grundy SM, Abate N, Chandalia M. Diet composition and the metabolic syndrome: what is the optimal
fat intake? Am J Med. 2002 Dec 30;113 Suppl 9B:25S-29S. Review. <ncbi.nlm.nih.goventrezOpen ↗
/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12566135&dopt=Abstract> ( tinyurl.comwaq1Open ↗ )
"... Emerging data suggest that diets higher in unsaturated fatty acids, particularly
monounsaturated fatty acids, have several advantages over high-carbohydrate intakes."
[...]
At the same time, the experience with the Mediterranean population reveals that in healthier
populations, diets relatively high in unsaturated fatty acids are well tolerated and are
associated with a low prevalence of both coronary heart disease and type 2 diabetes."
Hu FB, van Dam RM, Liu S. Diet and risk of Type II diabetes: the role of types of fat and
carbohydrate. Diabetologia. 2001 Jul;44(7):805-17. Review. <ncbi.nlm.nih.govquerOpen ↗
y.fcgi?cmd=Retrieve&db=PubMed&list_uids=11508264&dopt=Abstract> ( tinyurl.comwat2Open ↗ )
"Although diet and nutrition are widely believed to play an important part in the development of
Type II (non-insulin- dependent) diabetes mellitus, specific dietary factors have not been
clearly defined. Much controversy exists about the relations between the amount and types of
dietary fat and carbohydrate and the risk of diabetes. In this article, we review in detail the
current evidence regarding the associations between different types of fats and carbohydrates
and insulin resistance and Type II diabetes. Our findings indicate that a higher intake of
polyunsaturated fat and possibly long-chain n-3 fatty acids could be beneficial, whereas a
higher intake of saturated fat and trans-fat could adversely affect glucose metabolism and
insulin resistance. In dietary practice, exchanging nonhydrogenated polyunsaturated fat for
saturated and trans- fatty acids could appreciably reduce risk of Type II diabetes. In addition,
a low-glycaemic index diet with a higher amount of fiber and minimally processed whole grain
products reduces glycaemic and insulinaemic responses and lowers the risk of Type II diabetes.
Dietary recommendations to prevent Type II diabetes should focus more on the quality of fat and
carbohydrate in the diet than quantity alone, in addition to balancing total energy intake with
expenditure to avoid overweight and obesity."
Quoted message said:Quoted message said:Perhaps you should check the abstract
Yes, I have seen most, if not all of them, and the full papers, when they are free.
The clinical trials show that replacing saturated fat or trans fat with MUFA has some benefit in a
high risk population.Big deal. This does not indicate that 40% fat is in any way superior to a 24% or lower.
Hu FB, Stampfer MJ, Manson JE, Rimm E, Colditz GA, Rosner BA, Hennekens CH, Willett WC. Dietary fat
intake and the risk of coronary heart disease in women. N Engl J Med. 1997 Nov 20;337(21):1491-9.
PMID: 9366580 [PubMed - indexed for MEDLINE] ncbi.nlm.nih.govquery.fcgiOpen ↗
ieve&db=PubMed&list_uids=9366580&dopt=Abstract
suggests that replacing carbohydrates in the diet with either monounsaturated or polyunsaturated fat
is ssociated with reduced risk of CHD. A citation from the abstract:
"... RESULTS: Each increase of 5 percent of energy intake from saturated fat, as compared with
equivalent energy intake from carbohydrates, was associated with a 17 percent increase in the
risk of coronary disease (relative risk, 1.17; 95 percent confidence interval, 0.97 to 1.41;
P=0.10). As compared with equivalent energy from carbohydrates, the relative risk for a 2
percent increment in energy intake from trans unsaturated fat was 1.93 (95 percent confidence
interval, 1.43 to 2.61; P<0.001); that for a 5 percent increment in energy from monounsaturated
fat was 0.81 (95 percent confidence interval, 0.65 to 1.00; P=0.05); and that for a 5 percent
increment in energy from polyunsaturated fat was 0.62 (95 percent confidence interval, 0.46 to
0.85; P= 0.003). Total fat intake was not signficantly related to the risk of coronary disease
(for a 5 percent increase in energy from fat, the relative risk was 1.02; 95 percent confidence
interval, 0.97 to 1.07; P=0.55). ..."
See also
The Soft Science of Dietary Fat Gary Taubes Science, Vol 291, Number 5513, Mar 30, 2001 pp. 2536-
2545 Winner of the 2001 Science in Society Journalism Award
jacquigingras.comThe%20Soft%20Science%20of%20Dietary%20Fat.pdfOpen ↗ nasw.orgmemOpen ↗
maint/awards/The%20soft%20science.pdf countcarbs.comsoftscience part1.htmOpen ↗
timinvermont.comfat.htmOpen ↗
Martinez JA, Corbalan MS, Sanchez-Villegas A, Forga L, Marti A, Martinez-Gonzalez MA. Obesity risk
is associated with carbohydrate intake in women carrying the Gln27Glu beta2-adrenoceptor
polymorphism. J Nutr. 2003 Aug;133(8):2549-54. PMID: 12888635 [PubMed - in process]
nutrition.org2549Open ↗ <ncbi.nlm.nih.govquery.Open ↗
fcgi?cmd=Retrieve&db=PubMed&list_uids=12888635&dopt=Abstract>
Scientists, Physicians Discuss Latest Discoveries for Stopping Cancer before It Starts:
aacr.org1132am.aspOpen ↗
"A low-carbohydrate diet, rather than a low-fat diet, may halt the progression of prostate
cancer. Conversely, vitamin A and related compounds present in dairy products, beef fat and fish
oil, slow the growth of prostate tumors."
More about this on the web page
Low-carbs to fight prostate cancer? nutraingredientsusa.comnews.aspOpen ↗
--
Matti Narkia