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Antioxidants in brain injury

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30 May 2007
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  1. J Neurosci Res. 2007 May 17; [Epub ahead of print] Links
    Peroxynitrite-mediated oxidative damage to brain mitochondria:
    Protective effects of peroxynitrite scavengers.Singh IN, Sullivan PG,
    Hall ED.
    Spinal Cord & Brain Injury Research Center, University of Kentucky
    College of Medicine, Lexington.

    Peroxynitrite-mediated oxidative damage has been implicated in brain
    mitochondrial respiratory dysfunction after traumatic brain injury
    (TBI), which precedes the onset of neuronal loss. The aim of this
    study was to investigate the detrimental effects of the peroxynitrite
    donor SIN-1 (3-morpholinosydnonimine) on isolated brain mitochondria
    and to screen penicillamine, a stoichiometric (1:1) peroxynitrite-
    scavenging agent, and tempol, a catalytic scavenger of peroxynitrite-
    derived radicals, as antioxidant mitochondrial protectants. Exposure
    of the isolated mitochondria to SIN-1 caused a significant dose-
    dependent decrease in the respiratory control ratio and was
    accompanied by a significant increase in state II respiration,
    followed by significant decreases (P < 0.05) in states III and V.
    These functional alterations occurred together with significant
    increases in mitochondrial protein carbonyl (PC), lipid peroxidation-
    related 4-hydroxynonenal (4-HNE), and 3-nitrotyrosine (3-NT) content.
    Penicillamine hydrochloride (10 muM) partially but significantly (P <
    0.05) protected against SIN-1-induced decreases in states III and V.
    However, a 2.5 muM concentration of tempol was able to significantly
    antagonize a 4-fold molar excess (10 muM) concentration of SIN-1 as
    effectively as were higher tempol concentrations, consistent with the
    likelihood that tempol works by a catalytic mechanism. The protection
    of mitochondrial respiration by penicillamine and tempol occurred in
    parallel with attenuation of PC, 4-HNE, and 3-NT. These results
    indicate that SIN-1 causes mitochondrial oxidative damage and complex
    I dysfunction and that antioxidant compounds that target either
    peroxynitrite or its radicals may be effective mitochondrial
    protectants in the treatment of neural injury. (c) 2007 Wiley-Liss,
    Inc.

    PMID: 17510982 [PubMed - as supplied by publisher]

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